Upregulation of CKIP-1 inhibits high-glucose induced inflammation and oxidative stress in HRECs and attenuates diabetic retinopathy by modulating Nrf2/ARE signaling pathway: an in vitro study

标题
Upregulation of CKIP-1 inhibits high-glucose induced inflammation and oxidative stress in HRECs and attenuates diabetic retinopathy by modulating Nrf2/ARE signaling pathway: an in vitro study
作者
关键词
Diabetic retinopathy, Inflammation, Oxidative stress, <em class="EmphasisTypeItalic ">CKIP</em>-<em class="EmphasisTypeItalic ">1</em>, <em class="EmphasisTypeItalic ">Nrf2</em>/<em class="EmphasisTypeItalic ">ARE</em> signal pathway
出版物
Cell and Bioscience
Volume 9, Issue 1, Pages -
出版商
Springer Science and Business Media LLC
发表日期
2019-08-23
DOI
10.1186/s13578-019-0331-x

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