L-type Calcium Channels are Involved in Iron-induced Neurotoxicity in Primary Cultured Ventral Mesencephalon Neurons of Rats

Oxidative stress and neurodegeneration: the involvement of iron

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Systemic isradipine treatment diminishes calcium-dependent mitochondrial oxidant stress

(2018) Jaime N. Guzman et al.   JOURNAL OF CLINICAL INVESTIGATION

Iron, Dopamine, and α-Synuclein Interactions in at-Risk Dopaminergic Neurons in Parkinson’s Disease

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An unexpected improvement in spatial learning and memory ability in alpha-synuclein A53T transgenic mice

(2017) Qi Liu et al.   JOURNAL OF NEURAL TRANSMISSION

Region-Specific Iron Measured by MRI as a Biomarker for Parkinson’s Disease

(2017) Xiaojun Guan et al.   Neuroscience Bulletin

Mitochondrial Iron Metabolism and Its Role in Neurodegeneration

(2017) Maxx P. Horowitz et al.   JOURNAL OF ALZHEIMERS DISEASE

Orexin-A increases the activity of globus pallidus neurons in both normal and parkinsonian rats

(2016) Yan Xue et al.   EUROPEAN JOURNAL OF NEUROSCIENCE

Iron overload-induced calcium signals modulate mitochondrial fragmentation in HT-22 hippocampal neuron cells

(2016) Dong Gil Lee et al.   TOXICOLOGY

Identifying the Threshold of Iron Deficiency in the Central Nervous System of the Rat by the Auditory Brainstem Response

(2015) Allison R. Greminger et al.   ASN Neuro

Anti-oxidation and Antiapoptotic Effects of Chondroitin Sulfate on 6-Hydroxydopamine-Induced Injury Through the Up-Regulation of Nrf2 and Inhibition of Mitochondria-Mediated Pathway

(2015) Chuanxia Ju et al.   NEUROCHEMICAL RESEARCH

The role of L-type voltage-gated calcium channels Cav1.2 and Cav1.3 in normal and pathological brain function

(2014) Stefan M. Berger et al.   CELL AND TISSUE RESEARCH

Antihypertensive Agents and Risk of Parkinson's Disease: A Nationwide Cohort Study

(2014) Yen-Chieh Lee et al.   PLoS One

Thromboxane A2 Mediates Iron-Overload Cardiomyopathy in Mice Through Calcineurin-Nuclear Factor of Activated T Cells Signaling Pathway

(2013) Heng Lin et al.   CIRCULATION JOURNAL

Blocking of L-type calcium channels protects hippocampal and nigral neurons against iron neurotoxicity The role of L-type calcium channels in iron-induced neurotoxicity

(2013) M. Ömer Bostanci et al.   INTERNATIONAL JOURNAL OF NEUROSCIENCE

Nifedipine Prevents Iron Accumulation and Reverses Iron-Overload-Induced Dopamine Neuron Degeneration in the Substantia Nigra of Rats

(2012) ZeGang Ma et al.   NEUROTOXICITY RESEARCH

Iron MediatesN-Methyl-d-aspartate Receptor-dependent Stimulation of Calcium-induced Pathways and Hippocampal Synaptic Plasticity

(2011) Pablo Muñoz et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

The L-type channel antagonist isradipine is neuroprotective in a mouse model of Parkinson's disease

(2011) E. Ilijic et al.   NEUROBIOLOGY OF DISEASE

Differential Effect of Nimodipine in Attenuating Iron-Induced Toxicity in Brain- and Blood–Brain Barrier-Associated Cell Types

(2011) J. A. Lockman et al.   NEUROCHEMICAL RESEARCH

Iron handling in hippocampal neurons: activity-dependent iron entry and mitochondria-mediated neurotoxicity

(2010) Ilaria Pelizzoni et al.   AGING CELL

Calcium, cellular aging, and selective neuronal vulnerability in Parkinson's disease

(2010) D. James Surmeier et al.   CELL CALCIUM

Up-regulation of divalent metal transporter 1 in 6-hydroxydopamine intoxication is IRE/IRP dependent

(2010) Hong Jiang et al.   CELL RESEARCH

Robust Pacemaking in Substantia Nigra Dopaminergic Neurons

(2009) J. N. Guzman et al.   JOURNAL OF NEUROSCIENCE

Calcium homeostasis, selective vulnerability and Parkinson's disease

(2009) C. Savio Chan et al.   TRENDS IN NEUROSCIENCES

Mitochondrial DNA Damage in Iron Overload

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Up-regulation of divalent metal transporter 1 is involved in 1-methyl-4-phenylpyridinium (MPP+)-induced apoptosis in MES23.5 cells

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Midbrain iron content in early Parkinson disease: A potential biomarker of disease status

(2008) W. R. W. Martin et al.   NEUROLOGY

Over-expressed human divalent metal transporter 1 is involved in iron accumulation in MES23.5 cells

(2007) Hua-Min Xu et al.   NEUROCHEMISTRY INTERNATIONAL