Atg5-mediated autophagy deficiency in proximal tubules promotes cell cycle G2/M arrest and renal fibrosis

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Autophagy delays apoptosis in renal tubular epithelial cells in cisplatin cytotoxicity

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Autophagy is required for G1/G0quiescence in response to nitrogen starvation inSaccharomyces cerevisiae

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Autophagy in diabetic nephropathy

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Autophagy Regulates TGF-  Expression and Suppresses Kidney Fibrosis Induced by Unilateral Ureteral Obstruction

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Cell cycle control in the kidney

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Cell cycle arrest and the evolution of chronic kidney disease from acute kidney injury

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Autophagy

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ATG5 is induced by DNA-damaging agents and promotes mitotic catastrophe independent of autophagy

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The Role of Autophagy in the Pathogenesis of Diabetic Nephropathy

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Autophagy Guards Against Cisplatin-Induced Acute Kidney Injury

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Calpain, Atg5 and Bak play important roles in the crosstalk between apoptosis and autophagy induced by influx of extracellular calcium

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Autophagy plays a critical role in kidney tubule maintenance, aging and ischemia-reperfusion injury

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Autophagy Promotes Intracellular Degradation of Type I Collagen Induced by Transforming Growth Factor (TGF)-β1

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Liver Angiotensinogen Is the Primary Source of Renal Angiotensin II

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Autophagy in proximal tubules protects against acute kidney injury

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Atg7 Modulates p53 Activity to Regulate Cell Cycle and Survival During Metabolic Stress

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Autophagy Protects the Proximal Tubule from Degeneration and Acute Ischemic Injury

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Autophagy Is a Renoprotective Mechanism During in Vitro Hypoxia and in Vivo Ischemia-Reperfusion Injury

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Methods in Mammalian Autophagy Research

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Calorie restriction enhances cell adaptation to hypoxia through Sirt1-dependent mitochondrial autophagy in mouse aged kidney

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Autophagy influences glomerular disease susceptibility and maintains podocyte homeostasis in aging mice

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Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury

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Fate Tracing Reveals the Pericyte and Not Epithelial Origin of Myofibroblasts in Kidney Fibrosis

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Autophagy Suppresses Tumorigenesis through Elimination of p62

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