Dysregulated expression of ACTN4 contributes to endothelial cell injury via the activation of the p38-MAPK/p53 apoptosis pathway in preeclampsia

标题
Dysregulated expression of ACTN4 contributes to endothelial cell injury via the activation of the p38-MAPK/p53 apoptosis pathway in preeclampsia
作者
关键词
Preeclampsia, Endothelial cell dysfunction, Oxidative stress, Apoptosis, P38-MAPK/p53 pathway
出版物
JOURNAL OF PHYSIOLOGY AND BIOCHEMISTRY
Volume -, Issue -, Pages -
出版商
Springer Science and Business Media LLC
发表日期
2019-08-10
DOI
10.1007/s13105-019-00700-9

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