STAT3-induced SMYD3 transcription enhances chronic lymphocytic leukemia cell growth in vitro and in vivo
出版年份 2019 全文链接
标题
STAT3-induced SMYD3 transcription enhances chronic lymphocytic leukemia cell growth in vitro and in vivo
作者
关键词
Chronic lymphocytic leukemia, STAT3, SMYD3, Transcription
出版物
INFLAMMATION RESEARCH
Volume 68, Issue 9, Pages 739-749
出版商
Springer Science and Business Media LLC
发表日期
2019-06-19
DOI
10.1007/s00011-019-01257-5
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Combined treatment with sorafenib and silibinin synergistically targets both HCC cells and cancer stem cells by enhanced inhibition of the phosphorylation of STAT3/ERK/AKT
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- The STAT3 inhibitor WP1066 reverses the resistance of chronic lymphocytic leukemia cells to histone deacetylase inhibitors induced by interleukin-6
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- (2014) Yong Liu et al. MEDICAL ONCOLOGY
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- High expression of trimethylated histone H3 lysine 4 is associated with poor prognosis in hepatocellular carcinoma
- (2012) Chuanchao He et al. HUMAN PATHOLOGY
- Knockdown of SMYD3 by RNA interference inhibits cervical carcinoma cell growth and invasion in vitro
- (2011) Shu-zhen Wang et al. BMB Reports
- STAT3 is constitutively phosphorylated on serine 727 residues, binds DNA, and activates transcription in CLL cells
- (2010) I. Hazan-Halevy et al. BLOOD
- Persistently Activated Stat3 Maintains Constitutive NF-κB Activity in Tumors
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- IL-6 and Stat3 Are Required for Survival of Intestinal Epithelial Cells and Development of Colitis-Associated Cancer
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