Increasing cell permeability of N-acetylglucosamine via 6-acetylation enhances capacity to suppress T-helper 1 (TH1)/TH17 responses and autoimmunity

标题
Increasing cell permeability of N-acetylglucosamine via 6-acetylation enhances capacity to suppress T-helper 1 (TH1)/TH17 responses and autoimmunity
作者
关键词
T cells, Flow cytometry, Mouse models, Cell staining, Toxicity, Cell differentiation, Multiple sclerosis, Cell metabolism
出版物
PLoS One
Volume 14, Issue 3, Pages e0214253
出版商
Public Library of Science (PLoS)
发表日期
2019-03-27
DOI
10.1371/journal.pone.0214253

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