标题
Mechanisms of HGF/Met Signaling to Brk and Sam68 in Breast Cancer Progression
作者
关键词
-
出版物
Hormones & Cancer
Volume 3, Issue 1-2, Pages 14-25
出版商
Springer Nature
发表日期
2011-11-29
DOI
10.1007/s12672-011-0097-z
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Multiple Mutations and Bypass Mechanisms Can Contribute to Development of Acquired Resistance to MET Inhibitors
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- Structural Basis for Homodimerization of the Src-associated during Mitosis, 68-kDa Protein (Sam68) Qua1 Domain
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- Sam68 regulates EMT through alternative splicing–activated nonsense-mediated mRNA decay of the SF2/ASF proto-oncogene
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