标题
Loss of the mitochondrial kinase PINK1 does not alter platelet function
作者
关键词
-
出版物
Scientific Reports
Volume 8, Issue 1, Pages -
出版商
Springer Nature America, Inc
发表日期
2018-09-20
DOI
10.1038/s41598-018-32716-4
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Basal Mitophagy Occurs Independently of PINK1 in Mouse Tissues of High Metabolic Demand
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- Inducing mitophagy in diabetic platelets protects against severe oxidative stress
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- Hypoxic mitophagy regulates mitochondrial quality and platelet activation and determines severity of I/R heart injury
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- Elimination of paternal mitochondria in mouse embryos occurs through autophagic degradation dependent on PARKIN and MUL1
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- Phosphoproteomic screening identifies Rab GTPases as novel downstream targets of PINK1
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- Identification of roles for the SNARE-associated protein, SNAP29, in mouse platelets
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- Parkin is activated by PINK1-dependent phosphorylation of ubiquitin at Ser65
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- Loss of iron triggers PINK1/Parkin-independent mitophagy
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- RhoG Protein Regulates Platelet Granule Secretion and Thrombus Formation in Mice
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- Mitochondrial impairment increases FL-PINK1 levels by calcium-dependent gene expression
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- Absence of Platelet Phenotype in Mice Lacking the Motor Protein Myosin Va
- (2013) Matthew T. Harper et al. PLoS One
- Mitochondrial Calcium and Reactive Oxygen Species Regulate Agonist-Initiated Platelet Phosphatidylserine Exposure
- (2012) Hyo-Jung Choo et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Regulation of mitochondrial permeability transition pore by PINK1
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- PINK1 is activated by mitochondrial membrane potential depolarization and stimulates Parkin E3 ligase activity by phosphorylating Serine 65
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- Genome-wide RNA-seq analysis of human and mouse platelet transcriptomes
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- Depletion of PINK1 affects mitochondrial metabolism, calcium homeostasis and energy maintenance
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- Glucose and collagen regulate human platelet activity through aldose reductase induction of thromboxane
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- Mitochondrial membrane potential regulates PINK1 import and proteolytic destabilization by PARL
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- PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin
- (2010) Derek P. Narendra et al. PLOS BIOLOGY
- Modulation of calcium signalling by mitochondria
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- Two distinct pathways regulate platelet phosphatidylserine exposure and procoagulant function
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- Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission
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- Parkinson Phenotype in Aged PINK1-Deficient Mice Is Accompanied by Progressive Mitochondrial Dysfunction in Absence of Neurodegeneration
- (2009) Suzana Gispert et al. PLoS One
- Impaired Platelet Procoagulant Mechanisms in Patients with Bleeding Disorders
- (2009) Harvey Weiss SEMINARS IN THROMBOSIS AND HEMOSTASIS
- Key Role of Platelet Procoagulant Activity in Tissue Factor-and Collagen-Dependent Thrombus Formation in Arterioles and VenulesIn VivoDifferential Sensitivity to Thrombin Inhibition
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- PINK1 Is Necessary for Long Term Survival and Mitochondrial Function in Human Dopaminergic Neurons
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- Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress
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- The kinase domain of mitochondrial PINK1 faces the cytoplasm
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