Genetic Ablation of Fgf23 or Klotho Does not Modulate Experimental Heart Hypertrophy Induced by Pressure Overload
出版年份 2017 全文链接
标题
Genetic Ablation of Fgf23 or Klotho Does not Modulate Experimental Heart Hypertrophy Induced by Pressure Overload
作者
关键词
-
出版物
Scientific Reports
Volume 7, Issue 1, Pages -
出版商
Springer Nature
发表日期
2017-09-07
DOI
10.1038/s41598-017-10140-4
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Up-regulation of FGF23 release by aldosterone
- (2016) Bingbing Zhang et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Posttranslational processing of FGF23 in osteocytes during the osteoblast to osteocyte transition
- (2016) Hiroyuki Yamamoto et al. BONE
- Patients with FGF23-related hypophosphatemic rickets/osteomalacia do not present with left ventricular hypertrophy
- (2016) Yuichi Takashi et al. ENDOCRINE RESEARCH
- Use of serum fibroblast growth factor 23 vs. plasma B-type natriuretic peptide levels in assessing the pathophysiology of patients with heart failure
- (2016) Miki Imazu et al. HYPERTENSION RESEARCH
- Inflammation and functional iron deficiency regulate fibroblast growth factor 23 production
- (2016) Valentin David et al. KIDNEY INTERNATIONAL
- Spleen contributes significantly to increased circulating levels of fibroblast growth factor23 in response to lipopolysaccharide-induced inflammation
- (2016) Shweta Bansal et al. NEPHROLOGY DIALYSIS TRANSPLANTATION
- FGF23 promotes myocardial fibrosis in mice through activation of β-catenin
- (2016) Huixin Hao et al. Oncotarget
- Soluble klotho and mortality: The Ludwigshafen Risk and Cardiovascular Health Study
- (2015) Vincent M. Brandenburg et al. ATHEROSCLEROSIS
- Membrane-bound Klotho is not expressed endogenously in healthy or uraemic human vascular tissue
- (2015) Rik Mencke et al. CARDIOVASCULAR RESEARCH
- Activation of Cardiac Fibroblast Growth Factor Receptor 4 Causes Left Ventricular Hypertrophy
- (2015) Alexander Grabner et al. Cell Metabolism
- Molecular and structural transition mechanisms in long-term volume overload
- (2015) Belal A. Mohamed et al. EUROPEAN JOURNAL OF HEART FAILURE
- Experimental Myocardial Infarction Upregulates Circulating Fibroblast Growth Factor-23
- (2015) Olena Andrukhova et al. JOURNAL OF BONE AND MINERAL RESEARCH
- Induction of cardiac FGF23/FGFR4 expression is associated with left ventricular hypertrophy in patients with chronic kidney disease
- (2015) Maren Leifheit-Nestler et al. NEPHROLOGY DIALYSIS TRANSPLANTATION
- Volume Overload and Adverse Outcomes in Chronic Kidney Disease: Clinical Observational and Animal Studies
- (2015) S.-C. Hung et al. Journal of the American Heart Association
- Pathophysiological impact of serum fibroblast growth factor 23 in patients with nonischemic cardiac disease and early chronic kidney disease
- (2014) Miki Imazu et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- Fibroblast Growth Factor-23, Cardiovascular Prognosis, and Benefit of Angiotensin-Converting Enzyme Inhibition in Stable Ischemic Heart Disease
- (2014) Jacob A. Udell et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- Soluble Klotho Protects against Uremic Cardiomyopathy Independently of Fibroblast Growth Factor 23 and Phosphate
- (2014) J. Xie et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- FGF23 protein expression in coronary arteries is associated with impaired kidney function
- (2014) Natalie A. van Venrooij et al. NEPHROLOGY DIALYSIS TRANSPLANTATION
- Dynamic regulation of FGF23 by Fam20C phosphorylation, GalNAc-T3 glycosylation, and furin proteolysis
- (2014) V. S. Tagliabracci et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- FGF23 is a novel regulator of intracellular calcium and cardiac contractility in addition to cardiac hypertrophy
- (2013) Chad D. Touchberry et al. AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM
- Spironolactone ameliorates PIT1-dependent vascular osteoinduction in klotho-hypomorphic mice
- (2013) Jakob Voelkl et al. JOURNAL OF CLINICAL INVESTIGATION
- Renal Expression of FGF23 in Progressive Renal Disease of Diabetes and the Effect of Ace Inhibitor
- (2013) Cristina Zanchi et al. PLoS One
- Long-TermFgf23Deficiency Does Not Influence Aging, Glucose Homeostasis, or Fat Metabolism in Mice with a Nonfunctioning Vitamin D Receptor
- (2012) Carmen Streicher et al. ENDOCRINOLOGY
- Mechanism of FGF23 processing in fibrous dysplasia
- (2012) Nisan Bhattacharyya et al. JOURNAL OF BONE AND MINERAL RESEARCH
- Fibroblast Growth Factor-23 and Death, Heart Failure, and Cardiovascular Events in Community-Living Individuals
- (2012) Joachim H. Ix et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- Higher fibroblast growth factor-23 increases the risk of all-cause and cardiovascular mortality in the community
- (2012) Johan Ärnlöv et al. KIDNEY INTERNATIONAL
- Klotho Lacks a Vitamin D Independent Physiological Role in Glucose Homeostasis, Bone Turnover, and Steady-State PTH Secretion In Vivo
- (2012) René Anour et al. PLoS One
- Plasma fibroblast growth factor 23, parathyroid hormone, phosphorus, and risk of coronary heart disease
- (2011) Eric N. Taylor et al. AMERICAN HEART JOURNAL
- FGF23 induces left ventricular hypertrophy
- (2011) Christian Faul et al. JOURNAL OF CLINICAL INVESTIGATION
- Midwall Fibrosis Is an Independent Predictor of Mortality in Patients With Aortic Stenosis
- (2011) Marc R. Dweck et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- Differential Cardiac Remodeling in Preload Versus Afterload
- (2010) Karl Toischer et al. CIRCULATION
- Serum intact FGF23 associate with left ventricular mass, hypertrophy and geometry in an elderly population
- (2009) Majd A.I. Mirza et al. ATHEROSCLEROSIS
- Fibroblast Growth Factor 23 and Left Ventricular Hypertrophy in Chronic Kidney Disease
- (2009) Orlando M. Gutiérrez et al. CIRCULATION
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