High fat diet-induced TGF-β/Gbb signaling provokes insulin resistance through the tribbles expression
出版年份 2016 全文链接
标题
High fat diet-induced TGF-β/Gbb signaling provokes insulin resistance through the tribbles expression
作者
关键词
-
出版物
Scientific Reports
Volume 6, Issue 1, Pages -
出版商
Springer Nature
发表日期
2016-08-03
DOI
10.1038/srep30265
参考文献
相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。- Tribbles homologue 3 stimulates canonical TGF-β signalling to regulate fibroblast activation and tissue fibrosis
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- Strategies for exploring TGF-β signaling in Drosophila
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- Drosophila Tribbles Antagonizes Insulin Signaling-Mediated Growth and Metabolism via Interactions with Akt Kinase
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- Tribbles 3 mediates endoplasmic reticulum stress-induced insulin resistance in skeletal muscle
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- A Drosophila Model of High Sugar Diet-Induced Cardiomyopathy
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- Protection from Obesity and Diabetes by Blockade of TGF-β/Smad3 Signaling
- (2011) Hariom Yadav et al. Cell Metabolism
- Smad3 Deficiency in Mice Protects Against Insulin Resistance and Obesity Induced by a High-Fat Diet
- (2011) C. K. Tan et al. DIABETES
- A high-sugar diet produces obesity and insulin resistance in wild-type Drosophila
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- (2010) Ryan T. Birse et al. Cell Metabolism
- Aberrant hepatic TRIB3 gene expression in insulin-resistant obese humans
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- Association of serum bone morphogenetic protein 4 levels with obesity and metabolic syndrome in non-diabetic individuals
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- (2008) S. de Ferranti et al. CLINICAL CHEMISTRY
- Drosophila short neuropeptide F signalling regulates growth by ERK-mediated insulin signalling
- (2008) Kyu-Sun Lee et al. NATURE CELL BIOLOGY
- Adipocyte dysfunctions linking obesity to insulin resistance and type 2 diabetes
- (2008) Adilson Guilherme et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- Reduction of DILP2 in Drosophila Triages a Metabolic Phenotype from Lifespan Revealing Redundancy and Compensation among DILPs
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