miR-200b inhibits TGF-β1-induced epithelial-mesenchymal transition and promotes growth of intestinal epithelial cells
出版年份 2013 全文链接
标题
miR-200b inhibits TGF-β1-induced epithelial-mesenchymal transition and promotes growth of intestinal epithelial cells
作者
关键词
-
出版物
Cell Death & Disease
Volume 4, Issue 3, Pages e541-e541
出版商
Springer Nature
发表日期
2013-03-14
DOI
10.1038/cddis.2013.22
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Regulation of epithelial barrier function by the inflammatory bowel disease candidate gene, PTPN2
- (2012) Declan F. McCole Annals of the New York Academy of Sciences
- Requirement of Cellular Prion Protein for Intestinal Barrier Function and Mislocalization in Patients With Inflammatory Bowel Disease
- (2012) Constance S.V. Petit et al. GASTROENTEROLOGY
- Proinflammatory Cytokines in the Pathogenesis of Inflammatory Bowel Diseases
- (2011) Warren Strober et al. GASTROENTEROLOGY
- Autophagy, Microbial Sensing, Endoplasmic Reticulum Stress, and Epithelial Function in Inflammatory Bowel Disease
- (2011) Arthur Kaser et al. GASTROENTEROLOGY
- Farnesoid X receptor activation inhibits inflammation and preserves the intestinal barrier in inflammatory bowel disease
- (2011) R. M. Gadaleta et al. GUT
- MiR-200b is involved in Tgf-β signaling to regulate mammalian palate development
- (2011) Jeong-Oh Shin et al. HISTOCHEMISTRY AND CELL BIOLOGY
- Functional Cooperation between Snail1 and Twist in the Regulation ofZEB1Expression during Epithelial to Mesenchymal Transition
- (2011) Natàlia Dave et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- p53 regulates epithelial–mesenchymal transition through microRNAs targeting ZEB1 and ZEB2
- (2011) Taewan Kim et al. JOURNAL OF EXPERIMENTAL MEDICINE
- HSP70 decreases receptor-dependent phosphorylation of Smad2 and blocks TGF-β-induced epithelial-mesenchymal transition
- (2011) Yihao Li et al. Journal of Genetics and Genomics
- Genetics and pathogenesis of inflammatory bowel disease
- (2011) Bernard Khor et al. NATURE
- Snail and the microRNA-200 Family Act in Opposition to Regulate Epithelial-to-Mesenchymal Transition and Germ Layer Fate Restriction in Differentiating ESCs
- (2011) Jennifer G. Gill et al. STEM CELLS
- Potential role for SNAIL family transcription factors in the etiology of Crohnʼs disease-associated fistulae
- (2010) Michael Scharl et al. INFLAMMATORY BOWEL DISEASES
- Identification of Epithelial to Mesenchymal Transition as a Novel Source of Fibroblasts in Intestinal Fibrosis
- (2010) Sarah N. Flier et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Role of Smad2/3 and p38 MAP kinase in TGF-β1-induced epithelial-mesenchymal transition of pulmonary epithelial cells
- (2010) Irina Kolosova et al. JOURNAL OF CELLULAR PHYSIOLOGY
- MicroRNA-200b regulates cyclin D1 expression and promotes S-phase entry by targeting RND3 in HeLa cells
- (2010) Wei Xia et al. MOLECULAR AND CELLULAR BIOCHEMISTRY
- Genome-wide association identifies multiple ulcerative colitis susceptibility loci
- (2010) Dermot P B McGovern et al. NATURE GENETICS
- Therapeutic Options to Modulate Barrier Defects in Inflammatory Bowel Disease
- (2009) Nina A. Hering et al. DIGESTIVE DISEASES
- Many roads to maturity: microRNA biogenesis pathways and their regulation
- (2009) Julia Winter et al. NATURE CELL BIOLOGY
- IL-23 and Autoimmunity: New Insights into the Pathogenesis of Inflammatory Bowel Disease
- (2008) Clara Abraham et al. Annual Review of Medicine
- Evidence for a role of epithelial mesenchymal transition during pathogenesis of fistulae in Crohnʼs disease
- (2008) Frauke Bataille et al. INFLAMMATORY BOWEL DISEASES
- The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1
- (2008) Philip A. Gregory et al. NATURE CELL BIOLOGY
- Role of the intestinal barrier in inflammatory bowel disease
- (2008) Mike G Laukoetter et al. WORLD JOURNAL OF GASTROENTEROLOGY
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