Polarized but Differential Localization and Recruitment of STIM1, Orai1 and TRPC Channels in Secretory Cells
出版年份 2010 全文链接
标题
Polarized but Differential Localization and Recruitment of STIM1, Orai1 and TRPC Channels in Secretory Cells
作者
关键词
-
出版物
TRAFFIC
Volume 12, Issue 2, Pages 232-245
出版商
Wiley
发表日期
2010-11-07
DOI
10.1111/j.1600-0854.2010.01138.x
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- A minimal regulatory domain in the C terminus of STIM1 binds to and activates ORAI1 CRAC channels
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- STIM1 Clusters and Activates CRAC Channels via Direct Binding of a Cytosolic Domain to Orai1
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- Ribosome-free Terminals of Rough ER Allow Formation of STIM1 Puncta and Segregation of STIM1 from IP3 Receptors
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- An endoplasmic reticulum/plasma membrane junction: STIM1/Orai1/TRPCs
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- Deletion of TRPC3 in Mice Reduces Store-Operated Ca2+ Influx and the Severity of Acute Pancreatitis
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- ORAI1 deficiency and lack of store-operated Ca2+ entry cause immunodeficiency, myopathy, and ectodermal dysplasia
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- The Intracellular Loop of Orai1 Plays a Central Role in Fast Inactivation of Ca2+Release-activated Ca2+Channels
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- TRPC3 Regulates Agonist-stimulated Ca2+Mobilization by Mediating the Interaction between Type I Inositol 1,4,5-Trisphosphate Receptor, RACK1, and Orai1
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- SOAR and the polybasic STIM1 domains gate and regulate Orai channels
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- A role for Orai in TRPC-mediated Ca2+ entry suggests that a TRPC:Orai complex may mediate store and receptor operated Ca2+ entry
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- Reversible translocation of EYFP-tagged STIM1 is coupled to calcium influx in insulin secreting β-cells
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- Complex Actions of 2-Aminoethyldiphenyl Borate on Store-operated Calcium Entry
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- The Slc26a4 transporter functions as an electroneutral Cl−/I−/HCO3−exchanger: role of Slc26a4 and Slc26a6 in I−and HCO3−secretion and in regulation of CFTR in the parotid duct
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- 2-Aminoethoxydiphenyl borate directly facilitates and indirectly inhibits STIM1-dependent gating of CRAC channels
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- STIM1 Gates TRPC Channels, but Not Orai1, by Electrostatic Interaction
- (2008) Weizhong Zeng et al. MOLECULAR CELL
- Functional interactions among Orai1, TRPCs, and STIM1 suggest a STIM-regulated heteromeric Orai/TRPC model for SOCE/Icrac channels
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