标题
Emerging Anatomy of the BAP1 Tumor Suppressor System
作者
关键词
-
出版物
SCIENCE
Volume 337, Issue 6101, Pages 1463-1464
出版商
American Association for the Advancement of Science (AAAS)
发表日期
2012-09-21
DOI
10.1126/science.1228463
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- BAP1 loss defines a new class of renal cell carcinoma
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- Loss of the Tumor Suppressor BAP1 Causes Myeloid Transformation
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- The nuclear deubiquitinase BAP1 is commonly inactivated by somatic mutations and 3p21.1 losses in malignant pleural mesothelioma
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- Germline mutations in BAP1 predispose to melanocytic tumors
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- The Ubiquitin Carboxyl Hydrolase BAP1 Forms a Ternary Complex with YY1 and HCF-1 and Is a Critical Regulator of Gene Expression
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- Histone H2A deubiquitinase activity of the Polycomb repressive complex PR-DUB
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- Frequent Mutation of BAP1 in Metastasizing Uveal Melanomas
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- Defining the Human Deubiquitinating Enzyme Interaction Landscape
- (2009) Mathew E. Sowa et al. CELL
- The Deubiquitinating Enzyme BAP1 Regulates Cell Growth via Interaction with HCF-1
- (2009) Yuichi J. Machida et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Association of C-Terminal Ubiquitin Hydrolase BRCA1-Associated Protein 1 with Cell Cycle Regulator Host Cell Factor 1
- (2009) S. Misaghi et al. MOLECULAR AND CELLULAR BIOLOGY
- BRCA1-Associated Protein-1 Is a Tumor Suppressor that Requires Deubiquitinating Activity and Nuclear Localization
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