Deconstructing complexin function in activating and clamping Ca2+-triggered exocytosis by comparing knockout and knockdown phenotypes
出版年份 2013 全文链接
标题
Deconstructing complexin function in activating and clamping Ca2+-triggered exocytosis by comparing knockout and knockdown phenotypes
作者
关键词
-
出版物
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Volume 110, Issue 51, Pages 20777-20782
出版商
Proceedings of the National Academy of Sciences
发表日期
2013-12-03
DOI
10.1073/pnas.1321367110
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Complexin Activates Exocytosis of Distinct Secretory Vesicles Controlled by Different Synaptotagmins
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- Neurotransmitter Release: The Last Millisecond in the Life of a Synaptic Vesicle
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- Complexin Controls Spontaneous and Evoked Neurotransmitter Release by Regulating the Timing and Properties of Synaptotagmin Activity
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- C-Terminal Complexin Sequence Is Selectively Required for Clamping and Priming But Not for Ca2+ Triggering of Synaptic Exocytosis
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- Binding of the complexin N terminus to the SNARE complex potentiates synaptic-vesicle fusogenicity
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- Distinct Domains of Complexins Bind SNARE Complexes and Clamp Fusionin Vitro
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- For Better or for Worse: Complexins Regulate SNARE Function and Vesicle Fusion
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