The Effect of Hyperglycaemia on In Vitro Cytokine Production and Macrophage Infection with Mycobacterium tuberculosis
出版年份 2015 全文链接
标题
The Effect of Hyperglycaemia on In Vitro Cytokine Production and Macrophage Infection with Mycobacterium tuberculosis
作者
关键词
Macrophages, Cytokines, Glucose, Mycobacterium tuberculosis, Diabetes mellitus, Cell differentiation, Tuberculosis, Phagocytosis
出版物
PLoS One
Volume 10, Issue 2, Pages e0117941
出版商
Public Library of Science (PLoS)
发表日期
2015-02-10
DOI
10.1371/journal.pone.0117941
参考文献
相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。- Diabetes and immunity to tuberculosis
- (2014) Nuria Martinez et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Phagocytosis via Complement or Fc-Gamma Receptors Is Compromised in Monocytes from Type 2 Diabetes Patients with Chronic Hyperglycemia
- (2014) Blanca I. Restrepo et al. PLoS One
- Host-pathogen interactions in tuberculosis patients with type 2 diabetes mellitus
- (2014) Blanca I. Restrepo et al. TUBERCULOSIS
- TANDEM: understanding diabetes and tuberculosis
- (2014) Reinout van Crevel et al. Lancet Diabetes & Endocrinology
- The Immune Response in Tuberculosis
- (2013) Anne O'Garra et al. Annual Review of Immunology
- Insulin Inhibits IL-10-Mediated Regulatory T Cell Function: Implications for Obesity
- (2013) J. M. Han et al. JOURNAL OF IMMUNOLOGY
- Glutathione deficiency in type 2 diabetes impairs cytokine responses and control of intracellular bacteria
- (2012) Kai Soo Tan et al. JOURNAL OF CLINICAL INVESTIGATION
- Mycobacterium tuberculosis Modulates Macrophage Lipid-Sensing Nuclear Receptors PPAR and TR4 for Survival
- (2012) S. Mahajan et al. JOURNAL OF IMMUNOLOGY
- Functional interactions between the gut microbiota and host metabolism
- (2012) Valentina Tremaroli et al. NATURE
- New pathways of protective and pathological host defense to mycobacteria
- (2012) Tom H.M. Ottenhoff TRENDS IN MICROBIOLOGY
- Reduced Mycobacterium tuberculosis association with monocytes from diabetes patients that have poor glucose control
- (2012) Diana I. Gomez et al. TUBERCULOSIS
- The phenotype and functional alterations of macrophages in mice with hyperglycemia for long term
- (2011) Chenming Sun et al. JOURNAL OF CELLULAR PHYSIOLOGY
- Phagocytic Activity Is Impaired in Type 2 Diabetes Mellitus and Increases after Metabolic Improvement
- (2011) Albert Lecube et al. PLoS One
- Enhanced protection to Mycobacterium tuberculosis infection in IL-10-deficient mice is accompanied by early and enhanced Th1 responses in the lung
- (2010) Paul S. Redford et al. EUROPEAN JOURNAL OF IMMUNOLOGY
- Quantification of microsized fluorescent particles phagocytosis to a better knowledge of toxicity mechanisms
- (2010) L. Leclerc et al. INHALATION TOXICOLOGY
- Cardiovascular and demographic characteristics in whole blood and plasma donors: results from the Donor InSight study
- (2010) Femke Atsma et al. TRANSFUSION
- Implications of the global increase of diabetes for tuberculosis control and patient care
- (2010) Rovina Ruslami et al. TROPICAL MEDICINE & INTERNATIONAL HEALTH
- CD14+ Monocytes Are Vulnerable and Functionally Impaired Under Endoplasmic Reticulum Stress in Patients With Type 2 Diabetes
- (2009) T. Komura et al. DIABETES
- Diabetes is associated with increased sensitivity of alveolar macrophages to urban particulate matter exposure
- (2009) Yiqun Mo et al. TOXICOLOGY
- Tuberculosis in Poorly Controlled Type 2 Diabetes: Altered Cytokine Expression in Peripheral White Blood Cells
- (2008) Blanca I. Restrepo et al. CLINICAL INFECTIOUS DISEASES
- Higher Susceptibility of Type 1 Diabetic Rats to Mycobacterium tuberculosis Infection
- (2008) Isamu Sugawara et al. TOHOKU JOURNAL OF EXPERIMENTAL MEDICINE
- Diabetes Mellitus Increases the Risk of Active Tuberculosis: A Systematic Review of 13 Observational Studies
- (2008) Christie Y Jeon et al. PLOS MEDICINE
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