标题
IL22/IL-22R Pathway Induces Cell Survival in Human Glioblastoma Cells
作者
关键词
Apoptosis, Cell proliferation, Phosphorylation, Gene expression, Cytokines, STAT signaling, Cell signaling, Colorectal cancer
出版物
PLoS One
Volume 10, Issue 3, Pages e0119872
出版商
Public Library of Science (PLoS)
发表日期
2015-03-21
DOI
10.1371/journal.pone.0119872
参考文献
相关参考文献
注意：仅列出部分参考文献，下载原文获取全部文献信息。- IL-22 produced by cancer-associated fibroblasts promotes gastric cancer cell invasion via STAT3 and ERK signaling
- (2014) H Fukui et al. BRITISH JOURNAL OF CANCER
- Interleukin-22 promotes epithelial cell transformation and breast tumorigenesis via MAP3K8 activation
- (2014) K. Kim et al. CARCINOGENESIS
- The role of the IL-22/IL-22R1 axis in cancer
- (2014) Chrissie Lim et al. CYTOKINE & GROWTH FACTOR REVIEWS
- SOCS3-mediated regulation of inflammatory cytokines in PTEN and p53 inactivated triple negative breast cancer model
- (2014) G Kim et al. ONCOGENE
- Inhibition of Keratinocyte Differentiation by the Synergistic Effect of IL-17A, IL-22, IL-1α, TNFα and Oncostatin M
- (2014) Hanitriniaina Rabeony et al. PLoS One
- High Expression of Interleukin-22 and Its Receptor Predicts Poor Prognosis in Pancreatic Ductal Adenocarcinoma
- (2013) Zhang Wen et al. ANNALS OF SURGICAL ONCOLOGY
- IL-22 is related to development of human colon cancer by activation of STAT3
- (2013) Runqiu Jiang et al. BMC CANCER
- Elevated serum IL-22 levels correlate with chemoresistant condition of colorectal cancer
- (2013) Tingyu Wu et al. CLINICAL IMMUNOLOGY
- Therapeutic opportunities of the IL-22–IL-22R1 system
- (2013) Robert Sabat et al. NATURE REVIEWS DRUG DISCOVERY
- Differential Expression of IL-17, 22 and 23 in the Progression of Colorectal Cancer in Patients with K-ras Mutation: Ras Signal Inhibition and Crosstalk with GM-CSF and IFN-γ
- (2013) Savvas Petanidis et al. PLoS One
- Interleukin 22-producing CD4+ T cells in malignant pleural effusion
- (2012) Zhi-Jian Ye et al. CANCER LETTERS
- Pro-tumour activity of interleukin-22 in HPAFII human pancreatic cancer cells
- (2012) L. M. Curd et al. CLINICAL AND EXPERIMENTAL IMMUNOLOGY
- IL-22 induced cell proliferation is regulated by PI3K/Akt/mTOR signaling cascade
- (2012) Anupam Mitra et al. CYTOKINE
- Glycosylation-related gene expression is linked to differentiation status in glioblastomas undifferentiated cells
- (2011) Mathilde Cheray et al. CANCER LETTERS
- Understanding the role of cytokines in Glioblastoma Multiforme pathogenesis
- (2011) Vivian F. Zhu et al. CANCER LETTERS
- Interleukin-22 promotes human hepatocellular carcinoma by activation of STAT3
- (2011) Runqiu Jiang et al. HEPATOLOGY
- Interleukin-22 Suppresses the Growth of A498 Renal Cell Carcinoma Cells via Regulation of STAT1 Pathway
- (2011) Fengbo Zhang et al. PLoS One
- STAT3 is essential for the maintenance of neurosphere-initiating tumor cells in patients with glioblastomas: A potential for targeted therapy?
- (2010) Claire Villalva et al. INTERNATIONAL JOURNAL OF CANCER
- Skin Inflammation Induced by the Synergistic Action of IL-17A, IL-22, Oncostatin M, IL-1 , and TNF- Recapitulates Some Features of Psoriasis
- (2010) K. Guilloteau et al. JOURNAL OF IMMUNOLOGY
- IL-10 and TGF-β2 are overexpressed in tumor spheres cultured from human gliomas
- (2010) Bo Qiu et al. MOLECULAR BIOLOGY REPORTS
- The Presence of IL-17A and T Helper 17 Cells in Experimental Mouse Brain Tumors and Human Glioma
- (2010) Derek A. Wainwright et al. PLoS One
- New Activation Modus of STAT3
- (2009) Laure Dumoutier et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Antiapoptotic Activity of Autocrine Interleukin-22 and Therapeutic Effects of Interleukin-22-Small Interfering RNA on Human Lung Cancer Xenografts
- (2008) W. Zhang et al. CLINICAL CANCER RESEARCH
- Involvement of miltefosine-mediated ERK activation in glioma cell apoptosis through Fas regulation
- (2008) Richa Tewari et al. JOURNAL OF NEUROCHEMISTRY
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