A miR-200b/200c/429-Binding Site Polymorphism in the 3′ Untranslated Region of the AP-2α Gene Is Associated with Cisplatin Resistance

Overexpression of miR-200c Induces Chemoresistance in Esophageal Cancers Mediated Through Activation of the Akt Signaling Pathway

(2011) R. Hamano et al.   CLINICAL CANCER RESEARCH

Single-Nucleotide Polymorphisms Inside MicroRNA Target Sites Influence Tumor Susceptibility

(2010) Milena S. Nicoloso et al.   CANCER RESEARCH

The expression of the miRNA-200 family in endometrial endometrioid carcinoma

(2010) Jeong-Won Lee et al.   GYNECOLOGIC ONCOLOGY

Tumour growth and resistance to gemcitabine of pancreatic cancer cells are decreased by AP-2α overexpression

(2009) N Jonckheere et al.   BRITISH JOURNAL OF CANCER

A Risk Variant in an miR-125b Binding Site in BMPR1B Is Associated with Breast Cancer Pathogenesis

(2009) P. Saetrom et al.   CANCER RESEARCH

DIANA-microT web server: elucidating microRNA functions through target prediction

(2009) M. Maragkakis et al.   NUCLEIC ACIDS RESEARCH

MicroRNA polymorphisms: the future of pharmacogenomics, molecular epidemiology and individualized medicine

(2009) Prasun J Mishra et al.   PHARMACOGENOMICS

The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

(2008) S.-M. Park et al.   GENES & DEVELOPMENT

The AP-2  transcription factor is required for the ganglioside GM3-stimulated transcriptional regulation of a PTEN gene

(2008) H.-J. Choi et al.   GLYCOBIOLOGY