Podocyte-Specific Overexpression of Wild Type or Mutant Trpc6 in Mice Is Sufficient to Cause Glomerular Disease

TRPC6 mutations associated with focal segmental glomerulosclerosis cause constitutive activation of NFAT-dependent transcription

(2009) Johannes Schlöndorff et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

The spectrum of focal segmental glomerulosclerosis: new insights

(2009) Vivette D DʼAgati   CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION

Mutations in the formin gene INF2 cause focal segmental glomerulosclerosis

(2009) Elizabeth J Brown et al.   NATURE GENETICS

TRPC6 mutational analysis in a large cohort of patients with focal segmental glomerulosclerosis

(2009) S. Santin et al.   NEPHROLOGY DIALYSIS TRANSPLANTATION

A Novel TRPC6 Mutation That Causes Childhood FSGS

(2009) Saskia F. Heeringa et al.   PLoS One

Tetracycline-Inducible Gene Expression in Conditionally Immortalized Mouse Podocytes

(2008) Hiroshi Kajiyama et al.   AMERICAN JOURNAL OF NEPHROLOGY

MYH9 is a major-effect risk gene for focal segmental glomerulosclerosis

(2008) Jeffrey B Kopp et al.   NATURE GENETICS

The actin cytoskeleton of kidney podocytes is a direct target of the antiproteinuric effect of cyclosporine A

(2008) Christian Faul et al.   NATURE MEDICINE

Podocyte injury in focal segmental glomerulosclerosis: Lessons from animal models (a play in five acts)

(2007) V.D. D'Agati   KIDNEY INTERNATIONAL