Gingerol-Induced Hypoxia-Inducible Factor 1 Alpha Inhibits Human Prion Peptide-Mediated Neurotoxicity

Mitochondria-Targeted Antioxidants Protect Against Amyloid-β Toxicity in Alzheimer's Disease Neurons

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Autophagy induced by resveratrol prevents human prion protein-mediated neurotoxicity

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[6]-Gingerol attenuates β-amyloid-induced oxidative cell death via fortifying cellular antioxidant defense system

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Hypoxia-inducible factor-1 alpha regulates prion protein expression to protect against neuron cell damage

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Emodin and [6]-gingerol lessen hypoxia-induced embryotoxicities in cultured mouse whole embryos via upregulation of hypoxia-inducible factor 1α and intracellular superoxide dismutases

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Morphological and Functional Abnormalities in Mitochondria Associated with Synaptic Degeneration in Prion Disease

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Age-related alterations affect the susceptibility of mice to prion infection

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The induction of HIF-1 reduces astrocyte activation by amyloid beta peptide

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Inhibition of Prolyl Hydroxylase Protects against 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced Neurotoxicity

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Repetitive Hypoxic Preconditioning Attenuates Renal Ischemia/Reperfusion Induced Oxidative Injury via Upregulating HIF-1α–Dependent bcl-2 Signaling

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Synergistic anti-Parkinsonism activity of high doses of B vitamins in a chronic cellular model

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Overactivation of calcineurin induced by amyloid-beta and prion proteins

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Mitochondrial biology and oxidative stress in Parkinson disease pathogenesis

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