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Analysis of Gender Differences in Genetic Risk: Association of TNFAIP3 Polymorphism with Male Childhood-Onset Systemic Lupus Erythematosus in the Japanese Population

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Interleukin-1α controls allergic sensitization to inhaled house dust mite via the epithelial release of GM-CSF and IL-33

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Influenza infection in suckling mice expands an NKT cell subset that protects against airway hyperreactivity

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Enterocyte-specific A20 deficiency sensitizes to tumor necrosis factor–induced toxicity and experimental colitis

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Inflammatory dendritic cells—not basophils—are necessary and sufficient for induction of Th2 immunity to inhaled house dust mite allergen

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Innate immune responses of airway epithelium to house dust mite are mediated through β-glucan–dependent pathways

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Ubiquitin-Editing Enzyme A20 Promotes Tolerance to Lipopolysaccharide in Enterocytes

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House dust mite allergen induces asthma via Toll-like receptor 4 triggering of airway structural cells

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The ubiquitin-editing enzyme A20 (TNFAIP3) is a central regulator of immunopathology

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Multiple polymorphisms in the TNFAIP3 region are independently associated with systemic lupus erythematosus

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