IRF7-Dependent Type I Interferon Production Induces Lethal Immune-Mediated Disease in STAT1 Knockout Mice Infected with Lymphocytic Choriomeningitis Virus

Interferon regulatory factor 7 (IRF7) is required for the optimal initial control but not subsequent clearance of lymphocytic choriomeningitis virus infection in mice

(2013) Wen Li et al.   VIROLOGY

Mice Deficient in STAT1 but Not STAT2 or IRF9 Develop a Lethal CD4+ T-Cell-Mediated Disease following Infection with Lymphocytic Choriomeningitis Virus

(2012) M. J. Hofer et al.   JOURNAL OF VIROLOGY

Role of Interferon Regulatory Factor 7 in T Cell Responses during Acute Lymphocytic Choriomeningitis Virus Infection

(2012) S. Zhou et al.   JOURNAL OF VIROLOGY

Differential Impact of Interferon Regulatory Factor 7 in Initiation of the Type I Interferon Response in the Lymphocytic Choriomeningitis Virus-Infected Central Nervous System versus the Periphery

(2012) J. E. Christensen et al.   JOURNAL OF VIROLOGY

STAT2 Mediates Innate Immunity to Dengue Virus in the Absence of STAT1 via the Type I Interferon Receptor

(2011) Stuart T. Perry et al.   PLoS Pathogens

Induction and Inhibition of Type I Interferon Responses by Distinct Components of Lymphocytic Choriomeningitis Virus

(2010) S. Zhou et al.   JOURNAL OF VIROLOGY

Emerging Viral Infections of the Central Nervous System

(2009) Kenneth L. Tyler   ARCHIVES OF NEUROLOGY

IFR-9/STAT2 Functional Interaction Drives Retinoic Acid-Induced Gene G Expression Independently of STAT1

(2009) Y.-J. Lou et al.   CANCER RESEARCH

Hematopoietic cell-derived interferon controls viral replication and virus-induced disease

(2008) P. A. Lang et al.   BLOOD

Lymphocytic choriomeningitis infection of the central nervous system

(2008) Silvia et al.   Frontiers in Bioscience-Landmark

Interferon type I responses in primary and secondary infections

(2008) Mohammed Alsharifi et al.   IMMUNOLOGY AND CELL BIOLOGY

Myelomonocytic cell recruitment causes fatal CNS vascular injury during acute viral meningitis

(2008) Jiyun V. Kim et al.   NATURE