SLO3 auxiliary subunit LRRC52 controls gating of sperm KSPER currents and is critical for normal fertility
出版年份 2015 全文链接
标题
SLO3 auxiliary subunit LRRC52 controls gating of sperm KSPER currents and is critical for normal fertility
作者
关键词
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出版物
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Volume 112, Issue 8, Pages 2599-2604
出版商
Proceedings of the National Academy of Sciences
发表日期
2015-02-10
DOI
10.1073/pnas.1423869112
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- SLO3 K+Channels Control Calcium Entry through CATSPER Channels in Sperm
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- The Ca2+-activated K+ current of human sperm is mediated by Slo3
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- LRRC52 (leucine-rich-repeat-containing protein 52), a testis-specific auxiliary subunit of the alkalization-activated Slo3 channel
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- Acid Extrusion from Human Spermatozoa Is Mediated by Flagellar Voltage-Gated Proton Channel
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- Genetic male infertility and mutation of CATSPER ion channels
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- The SLO3 sperm-specific potassium channel plays a vital role in male fertility
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- LRRC26 auxiliary protein allows BK channel activation at resting voltage without calcium
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- Na,K-ATPase 4 isoform is essential for sperm fertility
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- Control of hyperactivation in sperm
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