标题
Nmnat2 protects cardiomyocytes from hypertrophy via activation of SIRT6
作者
关键词
-
出版物
FEBS LETTERS
Volume 586, Issue 6, Pages 866-874
出版商
Wiley
发表日期
2012-02-21
DOI
10.1016/j.febslet.2012.02.014
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Resveratrol attenuates doxorubicin-induced cardiomyocyte apoptosis in mice through SIRT1-mediated deacetylation of p53
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- Sirt1 acts in association with PPAR to protect the heart from hypertrophy, metabolic dysregulation, and inflammation
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- Fasting promotes the expression of SIRT1, an NAD+-dependent protein deacetylase, via activation of PPARα in mice
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- PARP-1 Expression in the Mouse is Controlled by an Autoregulatory Loop: PARP-1 Binding to an Upstream S/MAR Element and to a Novel Recognition Motif in its Promoter Suppresses Transcription
- (2009) Melita Vidaković et al. JOURNAL OF MOLECULAR BIOLOGY
- AMPK regulates energy expenditure by modulating NAD+ metabolism and SIRT1 activity
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- Effects of a Rho kinase inhibitor on pressure overload induced cardiac hypertrophy and associated diastolic dysfunction
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- Sirt1 protects the heart from aging and stress
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- Sirt7 Increases Stress Resistance of Cardiomyocytes and Prevents Apoptosis and Inflammatory Cardiomyopathy in Mice
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