Myocardial deletion of transcription factor CHF1/Hey2 results in altered myocyte action potential and mild conduction system expansion but does not alter conduction system function or promote spontaneous arrhythmias

An Optimized and Simplified System of Mouse Embryonic Stem Cell Cardiac Differentiation for the Assessment of Differentiation Modifiers

(2014) Matthew E. Hartman et al.   PLoS One

Common variants at SCN5A-SCN10A and HEY2 are associated with Brugada syndrome, a rare disease with high risk of sudden cardiac death

(2013) Connie R Bezzina et al.   NATURE GENETICS

Transcription factor CHF1/Hey2 regulates EC coupling and heart failure in mice through regulation of FKBP12.6

(2012) Yonggang Liu et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Myocardial Notch Signaling Reprograms Cardiomyocytes to a Conduction-Like Phenotype

(2012) Stacey Rentschler et al.   CIRCULATION

Brugada Syndrome

(2012) Yuka Mizusawa et al.   Circulation-Arrhythmia and Electrophysiology

Notch signaling regulates murine atrioventricular conduction and the formation of accessory pathways

(2011) Stacey Rentschler et al.   JOURNAL OF CLINICAL INVESTIGATION

Neuregulin/ErbB Signaling Regulates Cardiac Subtype Specification in Differentiating Human Embryonic Stem Cells

(2010) Wei-Zhong Zhu et al.   CIRCULATION RESEARCH

Transcription factor CHF1/Hey2 regulates coronary vascular maturation

(2010) Tokumitsu Watanabe et al.   MECHANISMS OF DEVELOPMENT

Cardiac Applications for Human Pluripotent Stem Cells

(2009) Yuji Shiba et al.   CURRENT PHARMACEUTICAL DESIGN

Local Control of Excitation-Contraction Coupling in Human Embryonic Stem Cell-Derived Cardiomyocytes

(2009) Wei-Zhong Zhu et al.   PLoS One

The multifaceted role of Notch in cardiac development and disease

(2007) Frances A. High et al.   NATURE REVIEWS GENETICS