标题
Rac1 - a novel regulator of contraction-stimulated glucose uptake in skeletal muscle
作者
关键词
-
出版物
EXPERIMENTAL PHYSIOLOGY
Volume 99, Issue 12, Pages 1574-1580
出版商
Wiley
发表日期
2014-09-20
DOI
10.1113/expphysiol.2014.079194
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- GARNL1, a major RalGAP α subunit in skeletal muscle, regulates insulin-stimulated RalA activation and GLUT4 trafficking via interaction with 14-3-3 proteins
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- AMPK controls exercise endurance, mitochondrial oxidative capacity, and skeletal muscle integrity
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- A critical role of the small GTPase Rac1 in Akt2-mediated GLUT4 translocation in mouse skeletal muscle
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- Wnt7a stimulates myogenic stem cell motility and engraftment resulting in improved muscle strength
- (2014) C. Florian Bentzinger et al. JOURNAL OF CELL BIOLOGY
- Contraction-stimulated glucose transport in muscle is controlled by AMPK and mechanical stress but not sarcoplasmatic reticulum Ca2+ release
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- Tiam-1, a GEF for Rac1, plays a critical role in metformin-mediated glucose uptake in C2C12 cells
- (2013) Ga Young You et al. CELLULAR SIGNALLING
- Akt and Rac1 signaling are jointly required for insulin-stimulated glucose uptake in skeletal muscle and downregulated in insulin resistance
- (2013) Lykke Sylow et al. CELLULAR SIGNALLING
- Rac1 Signaling Is Required for Insulin-Stimulated Glucose Uptake and Is Dysregulated in Insulin-Resistant Murine and Human Skeletal Muscle
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- Role of RalA downstream of Rac1 in insulin-dependent glucose uptake in muscle cells
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- Rac1 Is a Novel Regulator of Contraction-Stimulated Glucose Uptake in Skeletal Muscle
- (2012) L. Sylow et al. DIABETES
- Cardiac remodeling caused by transgenic overexpression of a corn Rac gene
- (2011) Mohammad T. Elnakish et al. AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY
- TRPC3-mediated Ca2+ influx contributes to Rac1-mediated production of reactive oxygen species in MLP-deficient mouse hearts
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- The small GTPase Rac1 is a novel binding partner of Bcl-2 and stabilizes its antiapoptotic activity
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- AMP-activated protein kinase (AMPK) 1 2 muscle null mice reveal an essential role for AMPK in maintaining mitochondrial content and glucose uptake during exercise
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- Crucial role of the small GTPase Rac1 in insulin-stimulated translocation of glucose transporter 4 to the mouse skeletal muscle sarcolemma
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- A myosin II ATPase inhibitor reduces force production, glucose transport, and phosphorylation of AMPK and TBC1D1 in electrically stimulated rat skeletal muscle
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- Genetic impairment of AMPKα2 signaling does not reduce muscle glucose uptake during treadmill exercise in mice
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- Skeletal muscle glucose uptake during contraction is regulated by nitric oxide and ROS independently of AMPK
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- Regulation of Rac1 by Simvastatin in Endothelial Cells
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- The Rho/Rac exchange factor Vav2 controls nitric oxide–dependent responses in mouse vascular smooth muscle cells
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- Stretch-stimulated glucose uptake in skeletal muscle is mediated by reactive oxygen species and p38 MAP-kinase
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- Rapid Activation of Rac GTPase in Living Cells by Force Is Independent of Src
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- Co-regulation of constitutive nitric oxide synthases and NADPH oxidase by the small GTPase Rac
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- The dynamics of the nitric oxide release-transient from stretched muscle cells
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- Retinoic Acid Leads to Cytoskeletal Rearrangement through AMPK-Rac1 and Stimulates Glucose Uptake through AMPK-p38 MAPK in Skeletal Muscle Cells
- (2008) Yun Mi Lee et al. JOURNAL OF BIOLOGICAL CHEMISTRY
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