标题
The cellular and molecular mechanisms for neutropenia in Barth syndrome
作者
关键词
-
出版物
EUROPEAN JOURNAL OF HAEMATOLOGY
Volume 88, Issue 3, Pages 195-209
出版商
Wiley
发表日期
2011-10-24
DOI
10.1111/j.1600-0609.2011.01725.x
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Tafazzin knockdown causes hypertrophy of neonatal ventricular myocytes
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- Characterization of a Transgenic Short Hairpin RNA-Induced Murine Model of Tafazzin Deficiency
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- Role of calcium-independent phospholipase A2 in the pathogenesis of Barth syndrome
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- Cardiolipin provides an essential activating platform for caspase-8 on mitochondria
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- Mitochondrial Membrane Potential in Human Neutrophils Is Maintained by Complex III Activity in the Absence of Supercomplex Organisation
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