标题
FoxO3A promotes metabolic adaptation to hypoxia by antagonizing Myc function
作者
关键词
-
出版物
EMBO JOURNAL
Volume 30, Issue 22, Pages 4554-4570
出版商
Wiley
发表日期
2011-09-13
DOI
10.1038/emboj.2011.323
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- The MK5/PRAK Kinase and Myc Form a Negative Feedback Loop that Is Disrupted during Colorectal Tumorigenesis
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- Hypoxia. 2. Hypoxia regulates cellular metabolism
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- FoxOs Enforce a Progression Checkpoint to Constrain mTORC1-Activated Renal Tumorigenesis
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- FOXO3 Modulates Endothelial Gene Expression and Function by Classical and Alternative Mechanisms
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- The Histone Demethylases JMJD1A and JMJD2B Are Transcriptional Targets of Hypoxia-inducible Factor HIF
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- FOXO3A Regulates Peroxiredoxin III Expression in Human Cardiac Fibroblasts
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- Mitochondrial Autophagy Is an HIF-1-dependent Adaptive Metabolic Response to Hypoxia
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- c-Myc represses FOXO3a-mediated transcription of the gene encoding the p27Kip1 cyclin dependent kinase inhibitor
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- The MYCN oncogene is a direct target of miR-34a
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- FOXO animal models reveal a variety of diverse roles for FOXO transcription factors
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- Global Identification of Myc Target Genes Reveals Its Direct Role in Mitochondrial Biogenesis and Its E-Box Usage In Vivo
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- Unravelling the tumor-suppressive functions of FOXO proteins
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