Complement-mediated chronic inflammation is associated with diabetic microvascular complication

C5L2 receptor disruption enhances the development of diet-induced insulin resistance in mice

(2012) Alexandre Fisette et al.   IMMUNOBIOLOGY

Complement component C5a Promotes Expression of IL-22 and IL-17 from Human T cells and its Implication in Age-related Macular Degeneration

(2011) Baoying Liu et al.   Journal of Translational Medicine

Interleukin-18 contributes more closely to the progression of diabetic nephropathy than other diabetic complications

(2010) Takayuki Fujita et al.   ACTA DIABETOLOGICA

The C3a Anaphylatoxin Receptor Is a Key Mediator of Insulin Resistance and Functions by Modulating Adipose Tissue Macrophage Infiltration and Activation

(2009) Y. Mamane et al.   DIABETES

Complement Factor H Is Expressed in Adipose Tissue in Association With Insulin Resistance

(2009) J. M. Moreno-Navarrete et al.   DIABETES

Markers of endothelial dysfunction and low-grade inflammation are associated in the offspring of type 2 diabetic subjects

(2007) Eija Ruotsalainen et al.   ATHEROSCLEROSIS