期刊
CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY
卷 39, 期 1, 页码 96-103出版社
WILEY
DOI: 10.1111/j.1440-1681.2011.05482.x
关键词
heat shock proteins; lymphocytes; macrophages; pressure natriuresis; T-cells
资金
- FONACYT [FC-2005000283]
- CONACYT [79661]
- National Institutes of Health National Heart Lung and Blood Institute (NHLBI) [HL-6860]
1. The present article reviews the role of immune-competent cells infiltrating the kidney and their association with oxidative stress and renal angiotensin activity in the development of salt-sensitive hypertension. 2. We discuss changes in the pressure-natriuresis relationship resulting from renal inflammation and its improvement resulting from immunosuppressive treatment. 3. The potential role of T-cell-driven reactivity in sustaining the renal inflammation is examined in the light of accumulating evidence of autoimmune mechanisms in experimental and clinical hypertension.
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