The IL-17A/IL-17RA Axis Plays a Proatherogenic Role via the Regulation of Aortic Myeloid Cell Recruitment
出版年份 2012 全文链接
标题
The IL-17A/IL-17RA Axis Plays a Proatherogenic Role via the Regulation of Aortic Myeloid Cell Recruitment
作者
关键词
-
出版物
CIRCULATION RESEARCH
Volume 110, Issue 5, Pages 675-687
出版商
Ovid Technologies (Wolters Kluwer Health)
发表日期
2012-02-04
DOI
10.1161/circresaha.111.261784
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Role of Interleukin 17 in Inflammation, Atherosclerosis, and Vascular Function in Apolipoprotein E–Deficient Mice
- (2011) Meena S. Madhur et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Interleukin-17A Deficiency Accelerates Unstable Atherosclerotic Plaque Formation in Apolipoprotein E-Deficient Mice
- (2011) Keiko Danzaki et al. ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY
- Inhibition of IL-17A in atherosclerosis
- (2011) Xiang Cheng et al. ATHEROSCLEROSIS
- Mycophenolate Mofetil Decreases Atherosclerotic Lesion Size by Depression of Aortic T-Lymphocyte and Interleukin-17–Mediated Macrophage Accumulation
- (2011) Sibylle von Vietinghoff et al. JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
- Current views on the functions of interleukin-17A-producing cells in atherosclerosis
- (2011) Matthew Butcher et al. THROMBOSIS AND HAEMOSTASIS
- Expression of IL-17A in human atherosclerotic lesions is associated with increased inflammation and plaque vulnerability
- (2010) Christian Erbel et al. BASIC RESEARCH IN CARDIOLOGY
- Blockade of Interleukin-17A Results in Reduced Atherosclerosis in Apolipoprotein E–Deficient Mice
- (2010) Emily Smith et al. CIRCULATION
- Hyperlipidemia-Triggered Neutrophilia Promotes Early Atherosclerosis
- (2010) Maik Drechsler et al. CIRCULATION
- Interleukin-17: friend or foe in atherosclerosis?
- (2010) Soraya Taleb et al. CURRENT OPINION IN LIPIDOLOGY
- IL-17 family member cytokines: Regulation and function in innate immunity
- (2010) Joseph M. Reynolds et al. CYTOKINE & GROWTH FACTOR REVIEWS
- IL-17A Is Proatherogenic in High-Fat Diet-Induced and Chlamydia pneumoniae Infection-Accelerated Atherosclerosis in Mice
- (2010) S. Chen et al. JOURNAL OF IMMUNOLOGY
- A Critical Function of Th17 Proinflammatory Cells in the Development of Atherosclerotic Plaque in Mice
- (2010) Q. Gao et al. JOURNAL OF IMMUNOLOGY
- Pro-Inflammatory Effects of Interleukin-17A on Vascular Smooth Muscle Cells Involve NAD(P)H- Oxidase Derived Reactive Oxygen Species
- (2010) Eweline Pietrowski et al. JOURNAL OF VASCULAR RESEARCH
- Immune and Inflammatory Mechanisms of Atherosclerosis
- (2009) Elena Galkina et al. Annual Review of Immunology
- Attenuated atherosclerosis upon IL-17R signaling disruption in LDLr deficient mice
- (2009) T. van Es et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- IL-17 Stimulates Migration of Carotid Artery Vascular Smooth Muscle Cells in an MMP-9 Dependent Manner via p38 MAPK and ERK1/2-Dependent NF-κB and AP-1 Activation
- (2009) Gao Cheng et al. CELLULAR AND MOLECULAR NEUROBIOLOGY
- Interleukin-17 and Interferon-γ Are Produced Concomitantly by Human Coronary Artery–Infiltrating T Cells and Act Synergistically on Vascular Smooth Muscle Cells
- (2009) Raymond E. Eid et al. CIRCULATION
- Loss of SOCS3 expression in T cells reveals a regulatory role for interleukin-17 in atherosclerosis
- (2009) Soraya Taleb et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Inhibition of IL-17A Attenuates Atherosclerotic Lesion Development in ApoE-Deficient Mice
- (2009) C. Erbel et al. JOURNAL OF IMMUNOLOGY
- IL-17 Induces Monocyte Migration in Rheumatoid Arthritis
- (2009) S. Shahrara et al. JOURNAL OF IMMUNOLOGY
- Interleukin (IL)-1 promotes allogeneic T cell intimal infiltration and IL-17 production in a model of human artery rejection
- (2008) Deepak A. Rao et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Induction and effector functions of TH17 cells
- (2008) Estelle Bettelli et al. NATURE
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started