Splice-variant changes of the CaV3.2 T-type calcium channel mediate voltage-dependent facilitation and associate with cardiac hypertrophy and development

CaV2.1 P/Q-type calcium channel alternative splicing affects the functional impact of familial hemiplegic migraine mutations: Implications for calcium channelopathies

(2010) Paul J. Adams et al.   Channels

Heart Failure–Associated Changes in RNA Splicing of Sarcomere Genes

(2010) Sek Won Kong et al.   Circulation-Cardiovascular Genetics

The Ca V 3.2 T-Type Ca 2+ Channel Is Required for Pressure Overload–Induced Cardiac Hypertrophy in Mice

(2009) Chien-Sung Chiang et al.   CIRCULATION RESEARCH

Excitation–contraction coupling changes during postnatal cardiac development

(2009) Andrew P. Ziman et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

A Cav3.2 T-Type Calcium Channel Point Mutation Has Splice-Variant-Specific Effects on Function and Segregates with Seizure Expression in a Polygenic Rat Model of Absence Epilepsy

(2009) K. L. Powell et al.   JOURNAL OF NEUROSCIENCE

Molecular alteration of Cav1.2 calcium channel in chronic myocardial infarction

(2009) Ping Liao et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

Genesis and Regulation of the Heart Automaticity

(2008) Matteo E. Mangoni et al.   PHYSIOLOGICAL REVIEWS

Regulation of neuronal T-type calcium channels

(2008) Mircea C. Iftinca et al.   TRENDS IN PHARMACOLOGICAL SCIENCES