RECQ1 is required for cellular resistance to replication stress and catalyzes strand exchange on stalled replication fork structures
出版年份 2012 全文链接
标题
RECQ1 is required for cellular resistance to replication stress and catalyzes strand exchange on stalled replication fork structures
作者
关键词
-
出版物
CELL CYCLE
Volume 11, Issue 22, Pages 4252-4265
出版商
Informa UK Limited
发表日期
2012-11-08
DOI
10.4161/cc.22581
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- DNA binding residues in the RQC domain of Werner protein are critical for its catalytic activities
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- Chronic activation of DNA damage signals causes tumor immune evasion in the chemoresistant niche
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- DrosophilaRecQ4 Has a 3′-5′ DNA Helicase Activity That Is Essential for Viability
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- Anticancer activity of RecQL1 helicase siRNA in mouse xenograft models
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- The Human RecQ Helicases, BLM and RECQ1, Display Distinct DNA Substrate Specificities
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- RECQ1 Possesses DNA Branch Migration Activity
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