Mitochondria induce oxidative stress, generation of reactive oxygen species and redox state unbalance of the eye lens leading to human cataract formation: disruption of redox lens organization by phospholipid hydroperoxides as a common basis for cataract

Reactive Oxygen Species and the Aging Eye

(2016) Mark A. Babizhayev et al.   AMERICAN JOURNAL OF THERAPEUTICS

Accumulation of lipid peroxidation products in human cataracts

(2009) Mark A. Babizhayev   ACTA OPHTHALMOLOGICA

Hyperoxia and Thyroxine Treatment and the Relationships between Reactive Oxygen Species Generation, Mitochondrial Membrane Potential, and Cardiolipin in Human Lens Epithelial Cell Cultures

(2009) Li Huang et al.   CURRENT EYE RESEARCH

Targeting Mitochondria

(2008) Adam T. Hoye et al.   ACCOUNTS OF CHEMICAL RESEARCH

Mitochondrial function and redox control in the aging eye: Role of MsrA and other repair systems in cataract and macular degenerations

(2008) Lisa A. Brennan et al.   EXPERIMENTAL EYE RESEARCH

Cardiolipin deficiency leads to decreased cardiolipin peroxidation and increased resistance of cells to apoptosis

(2008) Zhentai Huang et al.   FREE RADICAL BIOLOGY AND MEDICINE

Intracellular Effects of NSAIDs/ASA in Oxidatively Stressed Human Lens Epithelial Cells in Culture

(2008) A. Petersen et al.   OPHTHALMIC RESEARCH

Protein kinase C epsilon activates lens mitochondrial cytochrome c oxidase subunit IV during hypoxia

(2007) Michael Barnett et al.   EXPERIMENTAL EYE RESEARCH