期刊
CELL
卷 143, 期 6, 页码 861-863出版社
CELL PRESS
DOI: 10.1016/j.cell.2010.11.040
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资金
- NCI NIH HHS [R01 CA122617] Funding Source: Medline
An acute but transient response to insulin is essential for glucose homeostasis in mammals. Chakraborty et al. (2010) uncover a new feedback mechanism regulating insulin signaling. They show that the inositol pyrophosphate IP7, which is produced in response to insulin, inhibits the Akt kinase, a primary effector of insulin signaling.
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