Nitric oxide synthase 2 is required for conversion of pro-fibrogenic inflammatory CD133+ progenitors into F4/80+ macrophages in experimental autoimmune myocarditis

Nitric oxide and redox mechanisms in the immune response

(2011) David A. Wink et al.   JOURNAL OF LEUKOCYTE BIOLOGY

Alternative Activation of Macrophages: Mechanism and Functions

(2010) Siamon Gordon et al.   IMMUNITY

Mechanisms of Cardiac Fibrosis in Inflammatory Heart Disease

(2010) Gabriela Kania et al.   TRENDS IN CARDIOVASCULAR MEDICINE

Myeloid Differentiation Factor-88/Interleukin-1 Signaling Controls Cardiac Fibrosis and Heart Failure Progression in Inflammatory Dilated Cardiomyopathy

(2009) Przemyslaw Blyszczuk et al.   CIRCULATION RESEARCH

Heart-Infiltrating Prominin-1 + /CD133 + Progenitor Cells Represent the Cellular Source of Transforming Growth Factor β–Mediated Cardiac Fibrosis in Experimental Autoimmune Myocarditis

(2009) Gabriela Kania et al.   CIRCULATION RESEARCH

Differential Macrophage Polarization in Male and Female BALB/c Mice Infected With Coxsackievirus B3 Defines Susceptibility to Viral Myocarditis

(2009) Kang Li et al.   CIRCULATION RESEARCH

Alternatively activated macrophages in infection and autoimmunity

(2009) DeLisa Fairweather et al.   JOURNAL OF AUTOIMMUNITY

Autoimmune myocarditis: Past, present and future

(2009) Florian Leuschner et al.   JOURNAL OF AUTOIMMUNITY

Myocarditis: Infection Versus Autoimmunity

(2009) Noel R. Rose   JOURNAL OF CLINICAL IMMUNOLOGY

Interleukin-13 Protects Against Experimental Autoimmune Myocarditis by Regulating Macrophage Differentiation

(2008) Daniela Cihakova et al.   AMERICAN JOURNAL OF PATHOLOGY