MiR-124 targets Slug to regulate epithelial–mesenchymal transition and metastasis of breast cancer

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miR-124 suppresses multiple steps of breast cancer metastasis by targeting a cohort of pro-metastatic genes in vitro

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SLUG/SNAI2 and Tumor Necrosis Factor Generate Breast Cells With CD44+/CD24- Phenotype

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MicroRNA-124 suppresses oral squamous cell carcinoma motility by targeting ITGB1

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SLUG-induced Elevation of D1 Cyclin in Breast Cancer Cells through the Inhibition of Its Ubiquitination

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Expression of Slug Is Regulated by c-Myb and Is Required for Invasion and Bone Marrow Homing of Cancer Cells of Different Origin

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miR-124a Is Important for Migratory Cell Fate Transition During Gastrulation of Human Embryonic Stem Cells

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miR-124 and miR-203 are epigenetically silenced tumor-suppressive microRNAs in hepatocellular carcinoma

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p53 controls cancer cell invasion by inducing the MDM2-mediated degradation of Slug

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miR-124 regulates adult neurogenesis in the subventricular zone stem cell niche

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miR-124 and miR-137 inhibit proliferation of glioblastoma multiforme cells and induce differentiation of brain tumor stem cells

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A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

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The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

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The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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The microRNAs miR-373 and miR-520c promote tumour invasion and metastasis

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