Different incidences of epigenetic but not genetic abnormalities between Wilms tumors in Japanese and Caucasian children

Wt1 ablation and Igf2 upregulation in mice result in Wilms tumors with elevated ERK1/2 phosphorylation

(2010) Qianghua Hu et al.   JOURNAL OF CLINICAL INVESTIGATION

WNT/β-catenin pathway activation in Wilms tumors: A unifying mechanism with multiple entries?

(2009) Marie Corbin et al.   GENES CHROMOSOMES & CANCER

WTXinactivation is a frequent, but late event in Wilms tumors without apparent clinical impact

(2009) Jenny Wegert et al.   GENES CHROMOSOMES & CANCER

Canonical WNT signalling determines lineage specificity in Wilms tumour

(2009) R Fukuzawa et al.   ONCOGENE

The tumor suppressor WTX shuttles to the nucleus and modulates WT1 activity

(2009) M. N. Rivera et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Duplication of paternalIGF2 or loss of maternalIGF2 imprinting occurs in half of Wilms tumors with various structuralWT1 abnormalities

(2008) Masayuki Haruta et al.   GENES CHROMOSOMES & CANCER

Wilms tumor genetics: Mutations inWT1,WTX, andCTNNB1 account for only about one-third of tumors

(2008) E. Cristy Ruteshouser et al.   GENES CHROMOSOMES & CANCER

Different mechanisms cause imprinting defects at the IGF2/H19 locus in Beckwith-Wiedemann syndrome and Wilms' tumour

(2008) F. Cerrato et al.   HUMAN MOLECULAR GENETICS

Functional inactivation of the WTX gene is not a frequent event in Wilms’ tumors

(2008) D Perotti et al.   ONCOGENE