Tribbles in disease: Signaling pathways important for cellular function and neoplastic transformation
出版年份 2011 全文链接
标题
Tribbles in disease: Signaling pathways important for cellular function and neoplastic transformation
作者
关键词
-
出版物
CANCER SCIENCE
Volume 102, Issue 6, Pages 1115-1122
出版商
Wiley
发表日期
2011-02-22
DOI
10.1111/j.1349-7006.2011.01914.x
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Down syndrome and GATA1 mutations in transient abnormal myeloproliferative disorder: mutation classes correlate with progression to myeloid leukemia
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- Transformation by Tribbles homolog 2 (Trib2) requires both the Trib2 kinase domain and COP1 binding
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- The TRIB3 R84 variant is associated with increased carotid intima–media thickness in vivo and with enhanced MAPK signalling in human endothelial cells
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- miR-328 Functions as an RNA Decoy to Modulate hnRNP E2 Regulation of mRNA Translation in Leukemic Blasts
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- Control of Adipose Tissue Inflammation Through TRB1
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- Human Tribbles homolog 1 functions as a negative regulator of retinoic acid receptor
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- Mixed Lineage Kinase-3 Stabilizes and Functionally Cooperates with TRIBBLES-3 to Compromise Mitochondrial Integrity in Cytokine-induced Death of Pancreatic Beta Cells
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- The pseudokinase tribbles homolog 3 interacts with ATF4 to negatively regulate insulin exocytosis in human and mouse β cells
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- Trib1 is a lipid- and myocardial infarction–associated gene that regulates hepatic lipogenesis and VLDL production in mice
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- Elevated Tribbles homolog 2–specific antibody levels in narcolepsy patients
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- Gain of MYC underlies recurrent trisomy of the MYC chromosome in acute promyelocytic leukemia
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- The Amiloride Derivative Phenamil Attenuates Pulmonary Vascular Remodeling by Activating NFAT and the Bone Morphogenetic Protein Signaling Pathway
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- Human TRIB2 is a repressor of FOXO that contributes to the malignant phenotype of melanoma cells
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- Increased expression of the Akt/PKB inhibitor TRB3 in osteoarthritic chondrocytes inhibits insulin-like growth factor 1-mediated cell survival and proteoglycan synthesis
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- GATA-2 Reinforces Megakaryocyte Development in the Absence of GATA-1
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- The Small Molecule Phenamil Induces Osteoblast Differentiation and Mineralization
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- A retroviral mutagenesis screen reveals strong cooperation between Bcl11a overexpression and loss of the Nf1 tumor suppressor gene
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- LDL uptake by monocytes in response to inflammation is MAPK dependent but independent of tribbles protein expression
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- Tribbles-2 is a novel regulator of inflammatory activation of monocytes
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- TRB3 suppresses adipocyte differentiation by negatively regulating PPARγ transcriptional activity
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