Rosiglitazone via upregulation of Akt/eNOS pathways attenuates dysfunction of endothelial progenitor cells, induced by advanced glycation end products

标题
Rosiglitazone via upregulation of Akt/eNOS pathways attenuates dysfunction of endothelial progenitor cells, induced by advanced glycation end products
作者
关键词
-
出版物
BRITISH JOURNAL OF PHARMACOLOGY
Volume 158, Issue 8, Pages 1865-1873
出版商
Wiley
发表日期
2009-11-17
DOI
10.1111/j.1476-5381.2009.00450.x

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