Muscle histone deacetylase 4 upregulation in amyotrophic lateral sclerosis: potential role in reinnervation ability and disease progression

A Mutation Causes MuSK Reduced Sensitivity to Agrin and Congenital Myasthenia

(2013) Asma Ben Ammar et al.   PLoS One

Treatment with trichostatin A initiated after disease onset delays disease progression and increases survival in a mouse model of amyotrophic lateral sclerosis

(2011) Young-Eun Yoo et al.   EXPERIMENTAL NEUROLOGY

ALS patients with SOD1 mutations in Switzerland show very diverse phenotypes and extremely long survival: Table 1

(2011) M Weber et al.   JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY

Muscle Gene Expression Is a Marker of Amyotrophic Lateral Sclerosis Severity

(2011) Pierre-François Pradat et al.   Neurodegenerative Diseases

Patients who survive 5 years or more with ALS in Olmsted County, 1925-2004

(2010) F. J. Mateen et al.   JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY

Identification of an Agrin Mutation that Causes Congenital Myasthenia and Affects Synapse Function

(2009) Caroline Huzé et al.   AMERICAN JOURNAL OF HUMAN GENETICS

MicroRNA-206 Delays ALS Progression and Promotes Regeneration of Neuromuscular Synapses in Mice

(2009) A. H. Williams et al.   SCIENCE

The deacetylase HDAC4 controls myocyte enhancing factor-2-dependent structural gene expression in response to neural activity

(2008) Todd J. Cohen et al.   FASEB JOURNAL

Predictors of long survival in amyotrophic lateral sclerosis: A population-based study

(2007) Stefano Zoccolella et al.   JOURNAL OF THE NEUROLOGICAL SCIENCES