标题
MST1, a key player, in enhancing fast skeletal muscle atrophy
作者
关键词
MST1, muscle atrophy, FOXO3a
出版物
BMC BIOLOGY
Volume 11, Issue 1, Pages 12
出版商
Springer Nature
发表日期
2013-02-02
DOI
10.1186/1741-7007-11-12
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Lysine methylation of FOXO3 regulates oxidative stress-induced neuronal cell death
- (2012) Qi Xie et al. EMBO REPORTS
- p300 Acetyltransferase activity differentially regulates the localization and activity of the FOXO homologues in skeletal muscle
- (2011) Sarah M. Senf et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Posttranslational modifications control FoxO3 activity during denervation
- (2011) Enrico Bertaggia et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Altered REDD1, myostatin, and Akt/mTOR/FoxO/MAPK signaling in streptozotocin-induced diabetic muscle atrophy
- (2011) Juha J. Hulmi et al. AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM
- Inhibition of IkappaB kinase alpha (IKKα) or IKKbeta (IKKβ) plus forkhead box O (Foxo) abolishes skeletal muscle atrophy
- (2011) S.A. Reed et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Ryanodine Receptor Oxidation Causes Intracellular Calcium Leak and Muscle Weakness in Aging
- (2011) Daniel C. Andersson et al. Cell Metabolism
- Inhibition of FoxO transcriptional activity prevents muscle fiber atrophy during cachexia and induces hypertrophy
- (2011) Sarah A. Reed et al. FASEB JOURNAL
- The c-Abl-MST1 Signaling Pathway Mediates Oxidative Stress-Induced Neuronal Cell Death
- (2011) L. Xiao et al. JOURNAL OF NEUROSCIENCE
- Fiber Types in Mammalian Skeletal Muscles
- (2011) Stefano Schiaffino et al. PHYSIOLOGICAL REVIEWS
- Yap is a novel regulator of C2C12 myogenesis
- (2010) Kevin I. Watt et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Myogenin and Class II HDACs Control Neurogenic Muscle Atrophy by Inducing E3 Ubiquitin Ligases
- (2010) Viviana Moresi et al. CELL
- Autophagy in skeletal muscle
- (2010) Marco Sandri FEBS LETTERS
- Hippo signaling at a glance
- (2010) Bin Zhao et al. JOURNAL OF CELL SCIENCE
- Heart failure increases atrogin-1 and MuRF1 gene expression in skeletal muscle with fiber type-specific atrophy
- (2010) Robson Francisco Carvalho et al. JOURNAL OF MOLECULAR HISTOLOGY
- Muscle atrophy in cancer: a role for nutrition and exercise
- (2009) Marina Mourtzakis et al. Applied Physiology Nutrition and Metabolism
- Muscle atrophy in experimental cancer cachexia: Is the IGF-1 signaling pathway involved?
- (2009) Fabio Penna et al. INTERNATIONAL JOURNAL OF CANCER
- Deletion of a Genomic Segment Containing the Cardiac Troponin I Gene Knocks Down Expression of the Slow Troponin T Gene and Impairs Fatigue Tolerance of Diaphragm Muscle
- (2009) Han-Zhong Feng et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- c-Jun N-terminal Kinase Enhances MST1-mediated Pro-apoptotic Signaling through Phosphorylation at Serine 82
- (2009) Wenzhi Bi et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- MDM2 Acts Downstream of p53 as an E3 Ligase to Promote FOXO Ubiquitination and Degradation
- (2009) Wei Fu et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Phosphoinositide 3-Kinase/Akt Inhibits MST1-Mediated Pro-apoptotic Signaling through Phosphorylation of Threonine 120
- (2009) Zengqiang Yuan et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- During muscle atrophy, thick, but not thin, filament components are degraded by MuRF1-dependent ubiquitylation
- (2009) Shenhav Cohen et al. JOURNAL OF CELL BIOLOGY
- A Cell-Intrinsic Role for Mst1 in Regulating Thymocyte Egress
- (2009) Y. Dong et al. JOURNAL OF IMMUNOLOGY
- Phosphorylation of cardiac troponin I by mammalian sterile 20-like kinase 1
- (2008) Bei You et al. BIOCHEMICAL JOURNAL
- Does AMP-Activated Protein Kinase Negatively Mediate Aged Fast-Twitch Skeletal Muscle Mass?
- (2008) Scott E. Gordon et al. EXERCISE AND SPORT SCIENCES REVIEWS
- The FoxO code
- (2008) D R Calnan et al. ONCOGENE
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More