标题
Factor XII inhibition reduces thrombus formation in a primate thrombosis model
作者
关键词
-
出版物
BLOOD
Volume 123, Issue 11, Pages 1739-1746
出版商
American Society of Hematology
发表日期
2014-01-10
DOI
10.1182/blood-2013-04-499111
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Antithrombotic Effect of Antisense Factor XI Oligonucleotide Treatment in Primates
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- Red blood cells mediate the onset of thrombosis in the ferric chloride murine model
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- Factor XII promotes blood coagulation independent of factor XI in the presence of long-chain polyphosphates
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- A Sequential Mechanism for Exosite-mediated Factor IX Activation by Factor XIa
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- Selective Factor XIIa Inhibition Attenuates Silent Brain Ischemia
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- Murine prolylcarboxypeptidase depletion induces vascular dysfunction with hypertension and faster arterial thrombosis
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- Factor XIIa regulates the structure of the fibrin clot independently of thrombin generation through direct interaction with fibrin
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- Factor XIIa Inhibitor Recombinant Human Albumin Infestin-4 Abolishes Occlusive Arterial Thrombus Formation Without Affecting Bleeding
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- Intrinsic Coagulation Activation and the Risk of Arterial Thrombosis in Young Women
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- Laminin promotes coagulation and thrombus formation in a factor XII-dependent manner
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- Extracellular DNA traps promote thrombosis
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- Patients with severe factor XI deficiency have a reduced incidence of deep-vein thrombosis
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- Factor XI contributes to thrombin generation in the absence of factor XII
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- Platelet Polyphosphates Are Proinflammatory and Procoagulant Mediators In Vivo
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- Factor XI deficiency in humans
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- Prevention of vascular graft occlusion and thrombus-associated thrombin generation by inhibition of factor XI
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- Reduced incidence of ischemic stroke in patients with severe factor XI deficiency
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- Evolution of the contact phase of vertebrate blood coagulation
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