CX3CR1 reduces Ly6Chigh-monocyte motility within and release from the bone marrow after chemotherapy in mice
出版年份 2013 全文链接
标题
CX3CR1 reduces Ly6Chigh-monocyte motility within and release from the bone marrow after chemotherapy in mice
作者
关键词
-
出版物
BLOOD
Volume 122, Issue 5, Pages 674-683
出版商
American Society of Hematology
发表日期
2013-06-18
DOI
10.1182/blood-2013-01-480749
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Fate Mapping Reveals Origins and Dynamics of Monocytes and Tissue Macrophages under Homeostasis
- (2012) Simon Yona et al. IMMUNITY
- Rapid monocyte kinetics in acute myocardial infarction are sustained by extramedullary monocytopoiesis
- (2012) Florian Leuschner et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Origins of tumor-associated macrophages and neutrophils
- (2012) V. Cortez-Retamozo et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Therapeutic applications of macrophage colony-stimulating factor-1 (CSF-1) and antagonists of CSF-1 receptor (CSF-1R) signaling
- (2011) D. A. Hume et al. BLOOD
- In vivo structure/function and expression analysis of the CX3C chemokine fractalkine
- (2011) K.-W. Kim et al. BLOOD
- Extramedullary Hematopoiesis Generates Ly-6C high Monocytes That Infiltrate Atherosclerotic Lesions
- (2011) Clinton S. Robbins et al. CIRCULATION
- Bone Marrow Mesenchymal Stem and Progenitor Cells Induce Monocyte Emigration in Response to Circulating Toll-like Receptor Ligands
- (2011) Chao Shi et al. IMMUNITY
- Bone marrow CD169+macrophages promote the retention of hematopoietic stem and progenitor cells in the mesenchymal stem cell niche
- (2011) Andrew Chow et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Cyclophosphamide enhances immunity by modulating the balance of dendritic cell subsets in lymphoid organs
- (2010) T. Nakahara et al. BLOOD
- Chemorepulsion by blood S1P regulates osteoclast precursor mobilization and bone remodeling in vivo
- (2010) Masaru Ishii et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Successful Colon Cancer Eradication after Chemoimmunotherapy Is Associated with Profound Phenotypic Change of Intratumoral Myeloid Cells
- (2010) J. Medina-Echeverz et al. JOURNAL OF IMMUNOLOGY
- Cyclophosphamide Induces Dynamic Alterations in the Host Microenvironments Resulting in a Flt3 Ligand-Dependent Expansion of Dendritic Cells
- (2010) Mohamed L. Salem et al. JOURNAL OF IMMUNOLOGY
- Applications of myeloid-specific promoters in transgenic mice support in vivo imaging and functional genomics but do not support the concept of distinct macrophage and dendritic cell lineages or roles in immunity
- (2010) D. A. Hume JOURNAL OF LEUKOCYTE BIOLOGY
- Development of Monocytes, Macrophages, and Dendritic Cells
- (2010) F. Geissmann et al. SCIENCE
- Blood Monocytes: Development, Heterogeneity, and Relationship with Dendritic Cells
- (2009) Cedric Auffray et al. Annual Review of Immunology
- CX3CR1+CD115+CD135+common macrophage/DC precursors and the role of CX3CR1 in their response to inflammation
- (2009) Cedric Auffray et al. JOURNAL OF EXPERIMENTAL MEDICINE
- An engineered CX3CR1 antagonist endowed with anti-inflammatory activity
- (2009) Karim Dorgham et al. JOURNAL OF LEUKOCYTE BIOLOGY
- Sphingosine-1-phosphate mobilizes osteoclast precursors and regulates bone homeostasis
- (2009) Masaru Ishii et al. NATURE
- Identification of Splenic Reservoir Monocytes and Their Deployment to Inflammatory Sites
- (2009) F. K. Swirski et al. SCIENCE
- CX3CR1 is required for monocyte homeostasis and atherogenesis by promoting cell survival
- (2008) L. Landsman et al. BLOOD
- Functional Adhesiveness of the CX3CL1 Chemokine Requires Its Aggregation
- (2008) Patricia Hermand et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Differentiation and heterogeneity in the mononuclear phagocyte system
- (2008) D A Hume Mucosal Immunology
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