标题
WNK4 is the major WNK positively regulating NCC in the mouse kidney
作者
关键词
-
出版物
BIOSCIENCE REPORTS
Volume 34, Issue 3, Pages 195-205
出版商
Portland Press Ltd.
发表日期
2014-03-25
DOI
10.1042/bsr20140047
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Impaired KLHL3-Mediated Ubiquitination of WNK4 Causes Human Hypertension
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- Phosphorylation of Na–Cl cotransporter by OSR1 and SPAK kinases regulates its ubiquitination
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- Phosphatidylinositol 3-Kinase/Akt Signaling Pathway Activates the WNK-OSR1/SPAK-NCC Phosphorylation Cascade in Hyperinsulinemic db/db Mice
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- Overexpression of the Sodium Chloride Cotransporter Is Not Sufficient to Cause Familial Hyperkalemic Hypertension
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- Phenotypes of pseudohypoaldosteronism type II caused by the WNK4 D561A missense mutation are dependent on the WNK-OSR1/SPAK kinase cascade
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- The WNKs: Atypical Protein Kinases With Pleiotropic Actions
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- SPAK-Knockout Mice Manifest Gitelman Syndrome and Impaired Vasoconstriction
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- Targeted disruption of the Wnk4 gene decreases phosphorylation of Na-Cl cotransporter, increases Na excretion and lowers blood pressure
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