Effects of copper overload in P19 neurons: impairment of glutathione redox homeostasis and crosstalk between caspase and calpain protease systems in ROS-induced apoptosis

标题
Effects of copper overload in P19 neurons: impairment of glutathione redox homeostasis and crosstalk between caspase and calpain protease systems in ROS-induced apoptosis
作者
关键词
Copper toxicity, P19 neurons, Glutathione, Lactate dehydrogenase, Apoptosis, Calpain, ERK pathway
出版物
BIOMETALS
Volume 27, Issue 6, Pages 1303-1322
出版商
Springer Nature
发表日期
2014-09-13
DOI
10.1007/s10534-014-9792-x

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