Axonal autophagosomes recruit dynein for retrograde transport through fusion with late endosomes

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LC3 Binding to the Scaffolding Protein JIP1 Regulates Processive Dynein-Driven Transport of Autophagosomes

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The Regulation of Autophagosome Dynamics by Huntingtin and HAP1 Is Disrupted by Expression of Mutant Huntingtin, Leading to Defective Cargo Degradation

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Sugar modification inhibits autophagosome–lysosome fusion

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Snapin-Mediated BACE1 Retrograde Transport Is Essential for Its Degradation in Lysosomes and Regulation of APP Processing in Neurons

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Autophagosomal Syntaxin17-dependent lysosomal degradation maintains neuronal function inDrosophila

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Ordered Recruitment of Dynactin to the Microtubule Plus-End is Required for Efficient Initiation of Retrograde Axonal Transport

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Autophagosomes form at ER–mitochondria contact sites

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The Hairpin-type Tail-Anchored SNARE Syntaxin 17 Targets to Autophagosomes for Fusion with Endosomes/Lysosomes

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Autophagosomes initiate distally and mature during transport toward the cell soma in primary neurons

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Lysosomal Proteolysis Inhibition Selectively Disrupts Axonal Transport of Degradative Organelles and Causes an Alzheimer's-Like Axonal Dystrophy

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Huntingtin coordinates the dynein-mediated dynamic positioning of endosomes and lysosomes

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Snapin-Regulated Late Endosomal Transport Is Critical for Efficient Autophagy-Lysosomal Function in Neurons

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Dynein-dependent Movement of Autophagosomes Mediates Efficient Encounters with Lysosomes

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A neuron-specific cytoplasmic dynein isoform preferentially transports TrkB signaling endosomes

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The Itinerary of Autophagosomes: From Peripheral Formation to Kiss-and-Run Fusion with Lysosomes

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