The Type I BMP Receptor ACVR1/ALK2 is Required for Chondrogenesis During Development

Smad7 regulates terminal maturation of chondrocytes in the growth plate

(2013) Kristine D. Estrada et al.   DEVELOPMENTAL BIOLOGY

The face signature of fibrodysplasia ossificans progressiva

(2012) Peter Hammond et al.   AMERICAN JOURNAL OF MEDICAL GENETICS PART A

Fibrodysplasia ossificans progressiva: mechanisms and models of skeletal metamorphosis

(2012) F. S. Kaplan et al.   Disease Models & Mechanisms

AnAcvr1R206H knock-in mouse has fibrodysplasia ossificans progressiva

(2012) Salin A Chakkalakal et al.   JOURNAL OF BONE AND MINERAL RESEARCH

Cell cycle arrest in node cells governs ciliogenesis at the node to break left-right symmetry

(2011) Y. Komatsu et al.   DEVELOPMENT

Smad6 is essential to limit BMP signaling during cartilage development

(2011) Kristine D Estrada et al.   JOURNAL OF BONE AND MINERAL RESEARCH

TGFβ/BMP Type I Receptors ALK1 and ALK2 Are Essential for BMP9-induced Osteogenic Signaling in Mesenchymal Stem Cells

(2010) Jinyong Luo et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Conversion of vascular endothelial cells into multipotent stem-like cells

(2010) Damian Medici et al.   NATURE MEDICINE

BMP canonical Smad signaling through Smad1 and Smad5 is required for endochondral bone formation

(2009) K. N. Retting et al.   DEVELOPMENT

The type I BMP receptors, Bmpr1a and Acvr1, activate multiple signaling pathways to regulate lens formation

(2009) Ramya Rajagopal et al.   DEVELOPMENTAL BIOLOGY

Classic and atypical fibrodysplasia ossificans progressiva (FOP) phenotypes are caused by mutations in the bone morphogenetic protein (BMP) type I receptor ACVR1

(2008) Frederick S. Kaplan et al.   HUMAN MUTATION

Transforming Growth Factor  -Induced Smad1/5 Phosphorylation in Epithelial Cells Is Mediated by Novel Receptor Complexes and Is Essential for Anchorage-Independent Growth

(2008) A. C. Daly et al.   MOLECULAR AND CELLULAR BIOLOGY