期刊
JOURNAL OF BIOLOGICAL CHEMISTRY
卷 290, 期 31, 页码 18962-18966出版社
AMER SOC BIOCHEMISTRY MOLECULAR BIOLOGY INC
DOI: 10.1074/jbc.R115.640193
关键词
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资金
- National Institutes of Health [HL92774]
- Investigators in the Pathogenesis of Infectious Disease Award from the Burroughs Wellcome Fund
Several independent studies have recently converged upon the conclusion that the human bacterial pathogen Mycobacterium tuberculosis encounters copper during infections. At least three independently regulated pathways respond to excess copper and are required for the full virulence of M. tuberculosis in animals. In this review, I will discuss the functions of the best-characterized copper-responsive proteins in M. tuberculosis, the potential sources of copper during an infection, and remaining questions about the interface between copper and tuberculosis.
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