A decreased n-6/n-3 ratio in the fat-1 mouse is associated with improved glucose tolerance
出版年份 2010 全文链接
标题
A decreased n-6/n-3 ratio in the fat-1 mouse is associated with improved glucose tolerance
作者
关键词
-
出版物
Applied Physiology Nutrition and Metabolism
Volume 35, Issue 5, Pages 699-706
出版商
Canadian Science Publishing
发表日期
2010-10-21
DOI
10.1139/h10-066
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- (2010) Jonathan P. Little et al. JOURNAL OF PHYSIOLOGY-LONDON
- The Fat-1 Mouse has Brain Docosahexaenoic Acid Levels Achievable Through Fish Oil Feeding
- (2010) Sarah K. Orr et al. NEUROCHEMICAL RESEARCH
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- (2010) Cynthia Boudrault et al. NEUROCHEMISTRY INTERNATIONAL
- In obese rat muscle transport of palmitate is increased and is channeled to triacylglycerol storage despite an increase in mitochondrial palmitate oxidation
- (2009) Graham P. Holloway et al. AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM
- FAT/CD36-null mice reveal that mitochondrial FAT/CD36 is required to upregulate mitochondrial fatty acid oxidation in contracting muscle
- (2009) Graham P. Holloway et al. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY
- Prevention of insulin resistance by n-3 polyunsaturated fatty acids
- (2009) Dawn Fedor et al. CURRENT OPINION IN CLINICAL NUTRITION AND METABOLIC CARE
- n-3 Fatty acids and rosiglitazone improve insulin sensitivity through additive stimulatory effects on muscle glycogen synthesis in mice fed a high-fat diet
- (2009) O. Kuda et al. DIABETOLOGIA
- Transgenic expression ofn-3 fatty acid desaturase (fat-1) in C57/BL6 mice: Effects on glucose homeostasis and body weight
- (2009) Shaonin Ji et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Are all n-3 polyunsaturated fatty acids created equal?
- (2009) Breanne M Anderson et al. Lipids in Health and Disease
- Adiponectin resistance precedes the accumulation of skeletal muscle lipids and insulin resistance in high-fat-fed rats
- (2008) Kerry L. Mullen et al. AMERICAN JOURNAL OF PHYSIOLOGY-REGULATORY INTEGRATIVE AND COMPARATIVE PHYSIOLOGY
- Overexpression of Carnitine Palmitoyltransferase-1 in Skeletal Muscle Is Sufficient to Enhance Fatty Acid Oxidation and Improve High-Fat Diet-Induced Insulin Resistance
- (2008) C. R. Bruce et al. DIABETES
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- (2008) Jonathan D. Schertzer et al. ENDOCRINOLOGY
- Seizure resistance in fat-1 transgenic mice endogenously synthesizing high levels of omega-3 polyunsaturated fatty acids
- (2008) Ameer Y. Taha et al. JOURNAL OF NEUROCHEMISTRY
- Femur EPA and DHA are correlated with femur biomechanical strength in young fat-1 mice
- (2008) Beatrice Y.Y. Lau et al. JOURNAL OF NUTRITIONAL BIOCHEMISTRY
- Rosiglitazone increases fatty acid oxidation and fatty acid translocase (FAT/CD36) but not carnitine palmitoyltransferase I in rat muscle mitochondria
- (2008) Carley R. Benton et al. JOURNAL OF PHYSIOLOGY-LONDON
- Lipid Mediators of Insulin Resistance
- (2008) William L. Holland et al. NUTRITION REVIEWS
- High-fat diets cause insulin resistance despite an increase in muscle mitochondria
- (2008) C. R. Hancock et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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