Loss of phosphatase and tensin homolog (PTEN) in myeloid cells controls inflammatory bone destruction by regulating the osteoclastogenic potential of myeloid cells

Osteoporosis and fragility fractures

(2011) Lídia Sànchez-Riera et al.   BEST PRACTICE & RESEARCH IN CLINICAL RHEUMATOLOGY

Akt Induces Osteoclast Differentiation through Regulating the GSK3 /NFATc1 Signaling Cascade

(2011) J. B. Moon et al.   JOURNAL OF IMMUNOLOGY

Antiinflammatory effects of tumor necrosis factor on hematopoietic cells in a murine model of erosive arthritis

(2010) Stephan Blüml et al.   ARTHRITIS AND RHEUMATISM

Activating transcription factor 4 regulates osteoclast differentiation in mice

(2010) Huiling Cao et al.   JOURNAL OF CLINICAL INVESTIGATION

Anti-inflammatory properties of the PI3K pathway are mediated by IL-10/DUSP regulation

(2010) Philipp Günzl et al.   JOURNAL OF LEUKOCYTE BIOLOGY

Ca2+-NFATc1 signaling is an essential axis of osteoclast differentiation

(2009) Takako Negishi-Koga et al.   IMMUNOLOGICAL REVIEWS

Estrogen-dependent and C-C chemokine receptor-2–dependent pathways determine osteoclast behavior in osteoporosis

(2009) Nikolaus B Binder et al.   NATURE MEDICINE

Recent advances in the genetic analysis of PTEN and PI3K innate immune properties

(2008) Philipp Günzl et al.   IMMUNOBIOLOGY

TLR4/CD14-mediated PI3K activation is an essential component of interferon-dependent VSV resistance in macrophages

(2008) Gernot Schabbauer et al.   MOLECULAR IMMUNOLOGY

PTEN functions to 'prioritize' chemotactic cues and prevent 'distraction' in migrating neutrophils

(2008) Bryan Heit et al.   NATURE IMMUNOLOGY